Publisher Correction: Group 3 innate lymphoid cells mediate early protective immunity against tuberculosis (Nature, (2019), 570, 7762, (528-532), 10.1038/s41586-019-1276-2);Group 3 innate lymphoid cells mediate early protective immunity against tuberculosis

Nature

Volumn 570, Issue 7762, 2019, Pages 528-532

  Ardain, Amanda ^a,b   Domingo Gonzalez, Racquel ^c   Das, Shibali ^c   Kazer, Samuel W ^d,e,f   Howard, Nicole C ^c   Singh, Alveera ^a,b   Ahmed, Mushtaq ^c   Nhamoyebonde, Shepherd ^a,b   Rangel Moreno, Javier ^g   Ogongo, Paul ^a,b,h   Lu, Lan ^c   Ramsuran, Duran ^b   de la Luz Garcia Hernandez, Maria ^g   K Ulland, Tyler ^c   Darby, Matthew ⁱ   Park, Eugene ^c   Karim, Farina ^a   Melocchi, Laura ^c   Madansein, Rajhmun ^j   Dullabh, Kaylesh Jay ^j   Dunlap, Micah ^c   Marin Agudelo, Nancy ^c   Ebihara, Takashi ^c   Ndung’u, Thumbi ^a,b   Kaushal, Deepak ^k   Pym, Alexander S ^a,b   Kolls, Jay K ^l   Steyn, Adrie ^a,b,m   Zúñiga, Joaquín ^n,o   Horsnell, William ^i,p   Yokoyama, Wayne M ^c   Shalek, Alex K ^d,e,f   Kløverpris, Henrik N ^a,b,q,r   Colonna, Marco ^g   Leslie, Alasdair ^a,b,r   Khader, Shabaana A ^c   more..

^a AFRICA HEALTH RESEARCH INSTITUTE   (South Africa)

^b UNIVERSITY OF KWAZULU NATAL   (South Africa)

^c WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

^d Massachusetts Institute of Technology   (United States)

^e MASSACHUSETTS INSTITUTE OF TECHNOLOGY   (United States)

^f BROAD INSTITUTE OF MIT AND HARVARD   (United States)

^g UNIVERSITY OF ROCHESTER MEDICAL CENTER   (United States)

^h INSTITUTE OF PRIMATE RESEARCH   (Kenya)

ⁱ UNIVERSITY OF CAPE TOWN   (South Africa)

^j UNIVERSITY OF KWAZULU NATAL   (South Africa)

^k TULANE NATIONAL PRIMATE RESEARCH CENTER   (United States)

^l TULANE UNIVERSITY HEALTH SCIENCES CENTER   (United States)

^m UNIVERSITY OF ALABAMA AT BIRMINGHAM   (United States)

ⁿ INSTITUTO NACIONAL DE ENFERMEDADES RESPIRATORIAS   (Mexico)

^o SCHOOL OF ENGINEERING AND SCIENCES   (Mexico)

^p UNIVERSITY OF BIRMINGHAM   (United Kingdom)

^q UNIVERSITY OF COPENHAGEN   (Denmark)

^r UNIVERSITY COLLEGE LONDON   (United Kingdom)

more..

Author keywords

[No Author keywords available]

Indexed keywords

CHEMOKINE RECEPTOR CXCR5; CXCL13 CHEMOKINE; INTERLEUKIN 17; INTERLEUKIN 23; CXCL13 PROTEIN, HUMAN; CXCL13 PROTEIN, MOUSE; CXCR5 PROTEIN, HUMAN; INTERLEUKIN DERIVATIVE; INTERLEUKIN-22; TRANSCRIPTOME;

ADULT; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; C57BL 6 MOUSE; COHORT ANALYSIS; CONTROLLED STUDY; CYTOKINE PRODUCTION; FEMALE; GRANULOMA; HISTOPATHOLOGY; HUMAN; HUMAN CELL; HUMAN TISSUE; IMMUNITY; INNATE IMMUNITY; LETTER; LUNG ALVEOLUS MACROPHAGE; LUNG TUBERCULOSIS; LYMPH FOLLICLE; LYMPHOID CELL; MAJOR CLINICAL STUDY; MALE; MOUSE; MYCOBACTERIUM TUBERCULOSIS; NONHUMAN; PRIORITY JOURNAL; PROTEIN BLOOD LEVEL; TRANSCRIPTION INITIATION; UPREGULATION; ANIMAL; CLASSIFICATION; GENETICS; IMMUNOLOGY; LUNG; LYMPHOCYTE; METABOLISM; MICROBIOLOGY; PATHOLOGY;

MUS; MYCOBACTERIUM TUBERCULOSIS;

ANIMALS; CHEMOKINE CXCL13; FEMALE; GRANULOMA; HUMANS; IMMUNITY, INNATE; INTERLEUKIN-17; INTERLEUKINS; LUNG; LYMPHOCYTES; MACROPHAGES, ALVEOLAR; MALE; MICE; MYCOBACTERIUM TUBERCULOSIS; RECEPTORS, CXCR5; TRANSCRIPTOME; TUBERCULOSIS, PULMONARY;

EID: 85066976219     PISSN: 00280836     EISSN: 14764687     Source Type: Journal    
DOI: 10.1038/s41586-019-1458-y     Document Type: Erratum

References (31)

1. World Health Organization. Global Tuberculosis Report 2018. (2018).
2. Klose, C. S. & Artis, D. Innate lymphoid cells as regulators of immunity, inflammation and tissue homeostasis. Nat. Immunol. 17, 765–774 (2016).
3. Diefenbach, A., Colonna, M. & Koyasu, S. Development, differentiation, and diversity of innate lymphoid cells. Immunity 41, 354–365 (2014).
4. Takatori, H. et al. Lymphoid tissue inducer-like cells are an innate source of IL-17 and IL-22. J. Exp. Med. 206, 35–41 (2009).
5. Jones, G. W. & Jones, S. A. Ectopic lymphoid follicles: inducible centres for generating antigen-specific immune responses within tissues. Immunology 147, 141–151 (2016).
6. Lim, A. I. et al. Systemic human ILC precursors provide a substrate for tissue ILC differentiation. Cell 168, 1086–1100 (2017).
7. Monticelli, L. A. et al. Innate lymphoid cells promote lung-tissue homeostasis after infection with influenza virus. Nat. Immunol. 12, 1045–1054 (2011).
8. Mchedlidze, T. et al. Interleukin-33-dependent innate lymphoid cells mediate hepatic fibrosis. Immunity 39, 357–371 (2013).
9. Kløverpris, H. N. et al. Innate lymphoid cells are depleted irreversibly during acute HIV-1 infection in the absence of viral suppression. Immunity 44, 391–405 (2016).
10. Hedfors, I. A. & Brinchmann, J. E. Long-term proliferation and survival of in vitro-activated T cells is dependent on interleukin-2 receptor signalling but not on the high-affinity IL-2R. Scand. J. Immunol. 58, 522–532 (2003).
11. + T helper cells mediate protective immunity against tuberculosis. J. Clin. Invest. 123, 712–726 (2013).
12. Constantinides, M. G., McDonald, B. D., Verhoef, P. A. & Bendelac, A. A committed precursor to innate lymphoid cells. Nature 508, 397–401 (2014).
13. + lymphoid cells. Nature 463, 540–544 (2010).
14. Dorhoi, A. et al. The adaptor molecule CARD9 is essential for tuberculosis control. J. Exp. Med. 207, 777–792 (2010).
15. Traber, K. E. et al. Induction of STAT3-dependent CXCL5 expression and neutrophil recruitment by oncostatin-M during pneumonia. Am. J. Respir. Cell Mol. Biol. 53, 479–488 (2015).
16. Nouailles, G. et al. CXCL5-secreting pulmonary epithelial cells drive destructive neutrophilic inflammation in tuberculosis. J. Clin. Invest. 124, 1268–1282 (2014).
17. Pagán, A. J. et al. Myeloid growth factors promote resistance to mycobacterial infection by curtailing granuloma necrosis through macrophage replenishment. Cell Host Microbe 18, 15–26 (2015).
18. van de Veerdonk, F. L. et al. Mycobacterium tuberculosis induces IL-17A responses through TLR4 and dectin-1 and is critically dependent on endogenous IL-1. J. Leukoc. Biol. 88, 227–232 (2010).
19. Yadav, M. & Schorey, J. S. The β-glucan receptor dectin-1 functions together with TLR2 to mediate macrophage activation by mycobacteria. Blood 108, 3168–3175 (2006).
20. El-Shazly, A. E. et al. Novel association between vasoactive intestinal peptide and CRTH2 receptor in recruiting eosinophils: a possible biochemical mechanism for allergic eosinophilic inflammation of the airways. J. Biol. Chem. 288, 1374–1384 (2013).
21. Rajaram, M. V. et al. Mycobacterium tuberculosis activates human macrophage peroxisome proliferator-activated receptor γ linking mannose receptor recognition to regulation of immune responses. J. Immunol. 185, 929–942 (2010).
22. Tientcheu, L. D. et al. Differential transcriptomic and metabolic profiles of M. africanum- and M. tuberculosis-infected patients after, but not before, drug treatment. Genes Immun. 16, 347–355 (2015).
23. O’Kane, C. M., Elkington, P. T. & Friedland, J. S. Monocyte-dependent oncostatin M and TNF-α synergize to stimulate unopposed matrix metalloproteinase-1/3 secretion from human lung fibroblasts in tuberculosis. Eur. J. Immunol. 38, 1321–1330 (2008).
24. + T cell responses after vaccination and during Mycobacterium tuberculosis challenge. Nat. Immunol. 8, 369–377 (2007).
25. Ebihara, T. et al. Runx3 specifies lineage commitment of innate lymphoid cells. Nat. Immunol. 16, 1124–1133 (2015).
26. + natural killer-like cells. Nat. Immunol. 10, 66–74 (2009).
27. Trapnell, C. et al. Differential gene and transcript expression analysis of RNA-seq experiments with TopHat and Cufflinks. Nat. Protoc. 7, 562–578 (2012).
28. Li, B. D. & Dewey, C. N. RSEM: accurate transcript quantification from RNA-seq data with or without a reference genome. BMC Bioinformatics 12, 323 (2011).
29. Love, M. I., Huber, W. & Anders, S. Moderated estimation of fold change and dispersion for RNA-seq data with DESeq2. Genome Biol. 15, 550 (2014).
30. Bindea, G. et al. ClueGO: a Cytoscape plug-in to decipher functionally grouped gene ontology and pathway annotation networks. Bioinformatics 25, 1091–1093 (2009).
31. Upreti, D., Pathak, A. & Kung, S. K. Development of a standardized flow cytometric method to conduct longitudinal analyses of intracellular CD3ζ expression in patients with head and neck cancer. Oncol. Lett. 11, 2199–2206 (2016).