Reversion of β25-35-amyloid peptide-induced amnesia by NMDA receptor-associated glycine site agonists

Brain Research

Volumn 731, Issue 1-2, 1996, Pages 249-253

  Maurice, Tangui ^a   Lockhart, Brian P ^a,c   Su, Tsung Ping ^b   Privat, Alain ^a  

^a INSERM   (France)

^b NATIONAL INSTITUTE ON DRUG ABUSE   (United States)

^c INSTITUT DE RECHERCHES INTERNATIONALES SERVIER   (France)

Author keywords

Amnesia; D Cycloserine; Milacemide; NMDA glycine site; Amyloid

Indexed keywords

AMYLOID BETA PROTEIN; CYCLOSERINE; GLYCINE; MILACEMIDE; N METHYL DEXTRO ASPARTIC ACID RECEPTOR; ACETAMIDE DERIVATIVE; ANTIMETABOLITE; MONOAMINE OXIDASE INHIBITOR; PEPTIDE FRAGMENT;

AMNESIA; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; AVOIDANCE BEHAVIOR; CONTROLLED STUDY; INTRACEREBROVENTRICULAR DRUG ADMINISTRATION; LEARNING; MALE; MOUSE; NONHUMAN; PRIORITY JOURNAL; ANIMAL; ANIMAL BEHAVIOR; BINDING SITE; CHEMICALLY INDUCED DISORDER; CHEMISTRY; DRUG EFFECT; DRUG POTENTIATION; PHYSIOLOGY;

ACETAMIDES; AMNESIA; AMYLOID BETA-PROTEIN; ANIMALS; ANTIMETABOLITES; AVOIDANCE LEARNING; BEHAVIOR, ANIMAL; BINDING SITES; CYCLOSERINE; GLYCINE; MALE; MICE; MONOAMINE OXIDASE INHIBITORS; PEPTIDE FRAGMENTS; RECEPTORS, N-METHYL-D-ASPARTATE;

EID: 85047677709     PISSN: 00068993     EISSN: None     Source Type: Journal    
DOI: 10.1016/0006-8993(96)00710-X     Document Type: Article

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