Chronic obstructive pulmonary disease: Breathing new life into old cardiovascular drugs?

Annals of the American Thoracic Society

Volumn 14, Issue 11, 2017, Pages 1718-

  Polverino, Francesca ^a   Celli, Bartolome R ^a   Owen, Caroline A ^a  

^a HARVARD MEDICAL SCHOOL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ADVANCED GLYCATION END PRODUCT RECEPTOR; ANGIOTENSIN RECEPTOR ANTAGONIST; CARDIOVASCULAR AGENT; DIPEPTIDYL CARBOXYPEPTIDASE INHIBITOR; ENALAPRIL; TRANSFORMING GROWTH FACTOR BETA; ANGIOTENSIN DERIVATIVE;

CHRONIC OBSTRUCTIVE LUNG DISEASE; DRUG USE; EMPHYSEMA; ENDOTHELIAL DYSFUNCTION; HUMAN; LETTER; TGF BETA SIGNALING; X-RAY COMPUTED TOMOGRAPHY; ATHEROSCLEROSIS; TOMOGRAPHY;

ANGIOTENSIN RECEPTOR ANTAGONISTS; ANGIOTENSINS; ATHEROSCLEROSIS; CARDIOVASCULAR AGENTS; EMPHYSEMA; HUMANS; PULMONARY DISEASE, CHRONIC OBSTRUCTIVE; TOMOGRAPHY;

EID: 85032837187     PISSN: 23296933     EISSN: 23256621     Source Type: Journal    
DOI: 10.1513/annalsats.201707-613le     Document Type: Letter

References (6)

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4. Petersen H, Sood A, Meek PM, Shen X, Cheng Y, Belinsky SA, Owen CA, Washko G, Pinto-Plata V, Kelly E, et al. Rapid lung function decline in smokers is a risk factor for COPD and is attenuated by angiotensin-converting enzyme inhibitor use. Chest 2014; 145:695-703.
5. Polverino F, Celli BR, Owen CA. Reply to cigarette smoke is an endothelial cell toxin. Am J Respir Crit Care Med [online ahead of print] 20 Jul 2017; DOI: 10. 1164/ rccm. 201707-1381LE.
6. Voelkel NF. Cigarette smoke is an endothelial cell toxin. Am J Respir Crit Care Med [online ahead of print] 20 Jul 2017; DOI: 10. 1164/ rccm. 201706-1123LE.