Outbreak of H7N8 low pathogenic avian influenza in commercial turkeys with spontaneous mutation to highly pathogenic avian influenza

Genome Announcements

Volumn 4, Issue 3, 2016, Pages

  Killian, Mary Lea ^a   Kim Torchetti, Mia ^a   Hines, Nichole ^a   Yingst, Sam ^b   DeLiberto, Thomas ^c   Lee, Dong Hun ^d  

^a National Veterinary Services Laboratories   (United States)

^b PURDUE UNIVERSITY   (United States)

^c NATIONAL WILDLIFE RESEARCH CENTER   (United States)

^d AGRICULTURAL RESEARCH SERVICE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

EID: 85001088875     PISSN: None     EISSN: 21698287     Source Type: Journal    
DOI: 10.1128/genomeA.00457-16     Document Type: Article

References (4)

1. USDA-APHIS. 2016. Epidemiologic and Other Analyses of Indiana HPAI/LPAI-Affected Poultry Flocks: March 4, 2016 Report. USDA: APHIS:VS:STAS: Center for Epidemiology and Animal Health, Fort Collins, CO.
2. Swayne D, Suarez D, Sims L. 2013. Influenza, p. 181-218. In Swayne DE, Glisson JR, McDougald LR, Nolan LK, Suarez DL, Nair V (ed), Diseases of poultry, 13th ed. Wiley-Blackwell, Ames, IA.
3. Pasick J, Handel K, Robinson J, Copps J, Ridd D, Hills K, Kehler H, Cottam-Birt C, Neufeld J, Berhane Y, Czub S. 2005. Intersegmental recombination between the haemagglutinin and matrix genes was responsible for the emergence of a highly pathogenic H7N3 avian influenza virus in British Columbia. J Gen Virol 86:727-731. http://dx.doi.org/10.1099/vir.0.80478-0.
4. Maurer-Stroh S, Lee RTC, Gunalan V, Eisenhaber F. 2013. The highly pathogenic H7N3 avian influenza strain from July 2012 in Mexico acquired an extended cleavage site through recombination with host 28S rRNA. Virol J 10:139. http://dx.doi.org/10.1186/1743-422X-10-139.