Reply: Perifascicular necrosis in anti-synthetase syndrome beyond anti-Jo-1

Brain

Volumn 139, Issue 9, 2016, Pages e51-

  Allenbach, Yves ^a   Hervier, Baptise ^a   Stenzel, Werner ^b   Benveniste, Olivier ^a  

^a SORBONNE UNIVERSITÉ   (France)

^b CHARITÉ UNIVERSITÄTSMEDIZIN BERLIN   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

AMINOACYLTRANSFERASE; HLA DR ANTIGEN; JO 1 ANTIBODY; ANTINUCLEAR ANTIBODY; AUTOANTIBODY; HISTIDINE TRANSFER RNA LIGASE; LIGASE;

ANTISYNTHETASE SYNDROME; DERMATOMYOSITIS; HUMAN; LETTER; MYOPATHY; MYOSITIS; PERIFASCICULAR NECROTIZING MYOPATHY; PHENOTYPE; POLYMYOSITIS; PRIORITY JOURNAL;

ANTIBODIES, ANTINUCLEAR; AUTOANTIBODIES; HISTIDINE-TRNA LIGASE; HUMANS; LIGASES; MYOSITIS;

EID: 84994817653     PISSN: 00068950     EISSN: 14602156     Source Type: Journal    
DOI: 10.1093/brain/aww126     Document Type: Letter

References (5)

1. Aouizerate J, De Antonio M, Bassez G, Gherardi RK, Berenbaum F, Guillevin L, et al. Myofiber HLA-DR expression is a distinctive biomarker for antisynthetase-Associated myopathy. Acta Neuropathol Commun 2014; 2: 154.
2. De Bleecker JL, De Paepe B, Aronica E, de Visser M, ENMC Myositis Muscle Biopsy Study Group, Amato A, et al. 205th ENMC International Workshop: pathology diagnosis of idiopathic inflammatory myopathies part II 28-30 March 2014, Naarden, The Netherlands. Neuromuscul Disord 2015; 25: 268-72.
3. Hervier B, Devilliers H, Stanciu R, Meyer A, Uzunhan Y, Masseau A, et al. Hierarchical cluster and survival analyses of antisynthetase syndrome: phenotype and outcome are correlated with anti-TRNA synthetase antibody specificity. Autoimmun Rev 2012; 12: 210-17.
4. Stenzel W, Preube C, Allenbach Y, Pehl D, Junckerstorff R, Heppner FL, et al. Nuclear actin aggregation is a hallmark of anti-synthetase syndrome-induced dysimmune myopathy. Neurology 2015; 84: 1346-54.
5. Uruha A, Suzuki S, Suzuki N, Nishino I. Perifascicular necrosis in antisynthetase syndrome beyond anti-Jo-1. Brain 2016; 139: e50.