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Volumn 101, Issue 1, 2016, Pages 193-206

Protective effect of nitric oxide in aristolochic acid-induced toxic acute kidney injury: an old friend with new assets

Author keywords

[No Author keywords available]

Indexed keywords

ARGININE; ARISTOLOCHIC ACID; CREATININE; DRINKING WATER; N ACETYL BETA GLUCOSAMINIDASE; NITRIC OXIDE; REACTIVE OXYGEN METABOLITE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE PHOSPHATE OXIDASE 2; SODIUM;

EID: 84978520979     PISSN: 09580670     EISSN: 1469445X     Source Type: Journal    
DOI: 10.1113/EP085333     Document Type: Article
Times cited : (35)

References (35)
  • 1
    • 84871766223 scopus 로고    scopus 로고
    • Chronic l-arginine treatment improves metabolic, cardiovascular and liver complications in diet-induced obesity in rats
    • Alam MA, Kauter K, Withers K, Sernia C & Brown L (2013). Chronic l-arginine treatment improves metabolic, cardiovascular and liver complications in diet-induced obesity in rats. Food Funct 4, 83–91.
    • (2013) Food Funct , vol.4 , pp. 83-91
    • Alam, M.A.1    Kauter, K.2    Withers, K.3    Sernia, C.4    Brown, L.5
  • 7
    • 41549102309 scopus 로고    scopus 로고
    • Aristolochic acid nephropathy: a worldwide problem
    • Debelle FD, Vanherweghem JL & Nortier JL (2008). Aristolochic acid nephropathy: a worldwide problem. Kidney Int 74, 158–169.
    • (2008) Kidney Int , vol.74 , pp. 158-169
    • Debelle, F.D.1    Vanherweghem, J.L.2    Nortier, J.L.3
  • 8
    • 33748155399 scopus 로고    scopus 로고
    • Dynamics of hyaluronan, CD44, and inflammatory cells in the rat kidney after ischemia/reperfusion injury
    • Declèves AE, Caron N, Nonclercq D, Legrand A, Toubeau G, Kramp R & Flamion B (2006). Dynamics of hyaluronan, CD44, and inflammatory cells in the rat kidney after ischemia/reperfusion injury. Int J Mol Med 18, 83–94.
    • (2006) Int J Mol Med , vol.18 , pp. 83-94
    • Declèves, A.E.1    Caron, N.2    Nonclercq, D.3    Legrand, A.4    Toubeau, G.5    Kramp, R.6    Flamion, B.7
  • 9
    • 0036189846 scopus 로고    scopus 로고
    • Nitric oxide in acute renal failure: NOS versus NOS
    • Goligorsky MS, Brodsky SV & Noiri E (2002). Nitric oxide in acute renal failure: NOS versus NOS. Kidney Int 61, 855–861.
    • (2002) Kidney Int , vol.61 , pp. 855-861
    • Goligorsky, M.S.1    Brodsky, S.V.2    Noiri, E.3
  • 12
    • 58849099597 scopus 로고    scopus 로고
    • Diminished NO generation by injured endothelium and loss of macula densa nNOS may contribute to sustained acute kidney injury after ischemia-reperfusion
    • Kwon O, Hong SM & Ramesh G (2009). Diminished NO generation by injured endothelium and loss of macula densa nNOS may contribute to sustained acute kidney injury after ischemia-reperfusion. Am J Physiol Renal Physiol 296, F25–F33.
    • (2009) Am J Physiol Renal Physiol , vol.296 , pp. F25-F33
    • Kwon, O.1    Hong, S.M.2    Ramesh, G.3
  • 15
    • 79952628187 scopus 로고    scopus 로고
    • Aristolochic acid I suppressed iNOS gene expression and NF-κB activation in stimulated macrophage cells
    • Liu MC, Lin TH, Wu TS, Yu FY, Lu CC & Liu BH (2011). Aristolochic acid I suppressed iNOS gene expression and NF-κB activation in stimulated macrophage cells. Toxicol Lett 202, 93–99.
    • (2011) Toxicol Lett , vol.202 , pp. 93-99
    • Liu, M.C.1    Lin, T.H.2    Wu, T.S.3    Yu, F.Y.4    Lu, C.C.5    Liu, B.H.6
  • 16
    • 0035105457 scopus 로고    scopus 로고
    • l-Arginine enhances aerobic exercise capacity in association with augmented nitric oxide production
    • Maxwell AJ, Ho HV, Le CQ, Lin PS, Bernstein D & Cooke JP (2001). l-Arginine enhances aerobic exercise capacity in association with augmented nitric oxide production. J Appl Physiol 90, 933–938.
    • (2001) J Appl Physiol , vol.90 , pp. 933-938
    • Maxwell, A.J.1    Ho, H.V.2    Le, C.Q.3    Lin, P.S.4    Bernstein, D.5    Cooke, J.P.6
  • 17
    • 0025049273 scopus 로고
    • The first Robert Furchgott lecture: from endothelium-dependent relaxation to the l-arginine:NO pathway
    • Moncada S (1990). The first Robert Furchgott lecture: from endothelium-dependent relaxation to the l-arginine:NO pathway. Blood Vessels 27, 208–217.
    • (1990) Blood Vessels , vol.27 , pp. 208-217
    • Moncada, S.1
  • 18
    • 33745699168 scopus 로고    scopus 로고
    • Nitric oxide in the kidney: functions and regulation of synthesis
    • Mount PF & Power DA (2006). Nitric oxide in the kidney: functions and regulation of synthesis. Acta Physiol (Oxf) 187, 433–446.
    • (2006) Acta Physiol (Oxf) , vol.187 , pp. 433-446
    • Mount, P.F.1    Power, D.A.2
  • 22
    • 51349140430 scopus 로고    scopus 로고
    • Nitric oxide and cardiovascular and renal effects
    • Raij L (2008). Nitric oxide and cardiovascular and renal effects. Osteoarthritis Cartilage 16 Suppl 2, S21–S26.
    • (2008) Osteoarthritis Cartilage , vol.16 , pp. S21-S26
    • Raij, L.1
  • 23
    • 57449084343 scopus 로고    scopus 로고
    • Exogenous L-arginine ameliorates angiotensin II-induced hypertension and renal damage in rats
    • Rajapakse NW, De Miguel C, Das S & Mattson DL (2008). Exogenous L-arginine ameliorates angiotensin II-induced hypertension and renal damage in rats. Hypertension 52, 1084–1090.
    • (2008) Hypertension , vol.52 , pp. 1084-1090
    • Rajapakse, N.W.1    De Miguel, C.2    Das, S.3    Mattson, D.L.4
  • 24
    • 84871774231 scopus 로고    scopus 로고
    • Role of cellular L-arginine uptake and nitric oxide production on renal blood flow and arterial pressure regulation
    • Rajapakse NW & Mattson DL (2013). Role of cellular L-arginine uptake and nitric oxide production on renal blood flow and arterial pressure regulation. Curr Opin Nephrol Hypertens 22, 45–50.
    • (2013) Curr Opin Nephrol Hypertens , vol.22 , pp. 45-50
    • Rajapakse, N.W.1    Mattson, D.L.2
  • 25
    • 0036175503 scopus 로고    scopus 로고
    • Mechanism by which superoxide potentiates tubuloglomerular feedback
    • Ren Y, Carretero OA & Garvin JL (2002). Mechanism by which superoxide potentiates tubuloglomerular feedback. Hypertension 39, 624–628.
    • (2002) Hypertension , vol.39 , pp. 624-628
    • Ren, Y.1    Carretero, O.A.2    Garvin, J.L.3
  • 26
    • 76749148111 scopus 로고    scopus 로고
    • Intrarenal artery superoxide is mainly NADPH oxidase-derived and modulates endothelium-dependent dilation in elderly patients
    • Schlüter T, Zimmermann U, Protzel C, Miehe B, Klebingat KJ, Rettig R & Grisk O (2010). Intrarenal artery superoxide is mainly NADPH oxidase-derived and modulates endothelium-dependent dilation in elderly patients. Cardiovasc Res 85, 814–824.
    • (2010) Cardiovasc Res , vol.85 , pp. 814-824
    • Schlüter, T.1    Zimmermann, U.2    Protzel, C.3    Miehe, B.4    Klebingat, K.J.5    Rettig, R.6    Grisk, O.7
  • 28
    • 84885054027 scopus 로고    scopus 로고
    • NADPH oxidases, reactive oxygen species, and the kidney: friend and foe
    • Sedeek M, Nasrallah R, Touyz RM & Hebert RL (2013). NADPH oxidases, reactive oxygen species, and the kidney: friend and foe. J Am Soc Nephrol 24, 1512–1518.
    • (2013) J Am Soc Nephrol , vol.24 , pp. 1512-1518
    • Sedeek, M.1    Nasrallah, R.2    Touyz, R.M.3    Hebert, R.L.4
  • 29
    • 84913596330 scopus 로고    scopus 로고
    • Hypoxia as a key player in the AKI-to-CKD transition
    • Tanaka S, Tanaka T & Nangaku M (2014). Hypoxia as a key player in the AKI-to-CKD transition. Am J Physiol Renal Physiol 307, F1187–F1195.
    • (2014) Am J Physiol Renal Physiol , vol.307 , pp. F1187-F1195
    • Tanaka, S.1    Tanaka, T.2    Nangaku, M.3
  • 30
    • 84893198225 scopus 로고    scopus 로고
    • Downregulation of connective tissue growth factor by LPS/IFN-γ-induced nitric oxide is reversed by aristolochic acid treatment in glomerular mesangial cells via STAT-1α and NF-βB signaling
    • Tsai KD, Chen W, Wang SH, Hsiao YW, Chi JY, Wu HY, Lee YJ, Wong HY, Tseng MJ & Lin TH (2014). Downregulation of connective tissue growth factor by LPS/IFN-γ-induced nitric oxide is reversed by aristolochic acid treatment in glomerular mesangial cells via STAT-1α and NF-βB signaling. Chem Biol Interact 210, 86–95.
    • (2014) Chem Biol Interact , vol.210 , pp. 86-95
    • Tsai, K.D.1    Chen, W.2    Wang, S.H.3    Hsiao, Y.W.4    Chi, J.Y.5    Wu, H.Y.6    Lee, Y.J.7    Wong, H.Y.8    Tseng, M.J.9    Lin, T.H.10
  • 33
    • 0002687731 scopus 로고    scopus 로고
    • Nitric oxide metabolism following unilateral renal ischemia/reperfusion injury in rats
    • Waz WR, Van Liew JB & Feld LG (1998). Nitric oxide metabolism following unilateral renal ischemia/reperfusion injury in rats. Pediatr Nephrol 12, 26–29.
    • (1998) Pediatr Nephrol , vol.12 , pp. 26-29
    • Waz, W.R.1    Van Liew, J.B.2    Feld, L.G.3
  • 34
    • 46349098555 scopus 로고    scopus 로고
    • Ischemic injury underlies the pathogenesis of aristolochic acid-induced acute kidney injury
    • Wen YJ, Qu L & Li XM (2008). Ischemic injury underlies the pathogenesis of aristolochic acid-induced acute kidney injury. Transl Res 152, 38–46.
    • (2008) Transl Res , vol.152 , pp. 38-46
    • Wen, Y.J.1    Qu, L.2    Li, X.M.3
  • 35
    • 84885320788 scopus 로고    scopus 로고
    • Progressive endothelin-1 gene activation initiates chronic/end-stage renal disease following experimental ischemic/reperfusion injury
    • Zager RA, Johnson AC, Andress D & Becker K (2013). Progressive endothelin-1 gene activation initiates chronic/end-stage renal disease following experimental ischemic/reperfusion injury. Kidney Int 84, 703–712.
    • (2013) Kidney Int , vol.84 , pp. 703-712
    • Zager, R.A.1    Johnson, A.C.2    Andress, D.3    Becker, K.4


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.