Ivacaftor-induced proteomic changes suggest monocyte defects may contribute to the pathogenesis of cystic fibrosis

American Journal of Respiratory Cell and Molecular Biology

Volumn 54, Issue 4, 2016, Pages 594-599

  Hisert, Katherine B ^a   Schoenfelt, Kelly Q ^b   Cooke, Gordon ^c   Grogan, Brenda ^d   Launspach, Jan L ^e   Gallagher, Charles G ^c,d   Donnelly, Seamas C ^f   Welsh, Michael J ^e   Singh, Pradeep K ^a   McKone, Edward F ^c,d   Becker, Lev ^b  

^a UNIVERSITY OF WASHINGTON   (United States)

^b UNIVERSITY OF CHICAGO   (United States)

^c UNIVERSITY COLLEGE DUBLIN   (Ireland)

^d ST VINCENT S UNIVERSITY HOSPITAL   (Ireland)

^e UNIVERSITY OF IOWA   (United States)

^f TRINITY COLLEGE DUBLIN   (Ireland)

Author keywords

[No Author keywords available]

Indexed keywords

C REACTIVE PROTEIN; GAMMA INTERFERON; IVACAFTOR; AMINOPHENOL DERIVATIVE; CYSTIC FIBROSIS TRANSMEMBRANE CONDUCTANCE REGULATOR; PROTEOME; QUINOLONE DERIVATIVE;

CELL MIGRATION; CYSTIC FIBROSIS; DRUG EXPOSURE; ENDOCYTOSIS; HUMAN; LETTER; MACROPHAGE; MONOCYTE; PATHOGENESIS; PROTEOMICS; ANIMAL; DRUG EFFECTS; IMMUNOLOGY; METABOLISM; PATHOLOGY;

AMINOPHENOLS; ANIMALS; CYSTIC FIBROSIS; CYSTIC FIBROSIS TRANSMEMBRANE CONDUCTANCE REGULATOR; HUMANS; MONOCYTES; PROTEOME; QUINOLONES;

EID: 84963784799     PISSN: 10441549     EISSN: 15354989     Source Type: Journal    
DOI: 10.1165/rcmb.2015-0322le     Document Type: Letter

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