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Volumn 22, Issue 4, 2016, Pages 267-269
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'Mitotherapy' for Heart Failure
a
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Author keywords
Aging; Heart; Pathway
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Indexed keywords
MITOCHONDRIAL DNA;
MITOCHONDRIAL ESCAPE 1 LIKE PROTEIN;
OPTIC ATROPHY 1 PROTEIN;
PROTEIN;
TROPONIN T;
UNCLASSIFIED DRUG;
ARTERY OCCLUSION;
AUTOPHAGY;
BIOENERGY;
CELL COUNT;
CELL DIVISION;
CELL FUNCTION;
CELL FUSION;
CELL METABOLISM;
CELL MOTILITY;
CELL ORGANELLE;
CELL REGENERATION;
CELL STRESS;
CELL STRUCTURE;
CELL SUBPOPULATION;
CELL SURVIVAL;
CONGESTIVE CARDIOMYOPATHY;
DNA MODIFICATION;
FATTY ACID OXIDATION;
GENE ACTIVATION;
GENE EXPRESSION;
GENE INACTIVATION;
GENE MUTATION;
GENE OVEREXPRESSION;
GENE REPRESSION;
GLUCOSE TRANSPORT;
HEART FAILURE;
HEART FUNCTION;
HEART HYPERTROPHY;
HEART INFARCTION;
HEART LEFT VENTRICLE HYPERTROPHY;
HEART MITOCHONDRION;
HEART MUSCLE CELL;
HEART OUTPUT;
HEART PROTECTION;
HUMAN;
METABOLISM;
MITOCHONDRIAL DYNAMICS;
MITOPHAGY;
MITOTHERAPY;
MOLECULAR THERAPY;
MUSCLE FIBER CONTRACTION;
NONHUMAN;
OXIDATIVE STRESS;
PATHOPHYSIOLOGY;
PROTEIN BLOOD LEVEL;
PROTEIN DEGRADATION;
PROTEIN EXPRESSION;
PROTEIN FUNCTION;
PROTEIN TARGETING;
REPERFUSION INJURY;
SENESCENCE;
SHORT SURVEY;
SIGNAL TRANSDUCTION;
TELOMERE SHORTENING;
ANIMAL;
CARDIAC MUSCLE;
FEMALE;
MALE;
ANIMALS;
FEMALE;
HEART FAILURE;
MALE;
MITOCHONDRIA, HEART;
MITOCHONDRIAL DEGRADATION;
MITOCHONDRIAL DYNAMICS;
MYOCARDIUM;
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EID: 84959462407
PISSN: 14714914
EISSN: 1471499X
Source Type: Journal
DOI: 10.1016/j.molmed.2016.02.007 Document Type: Short Survey |
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Times cited : (6)
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References (12)
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