Effects of Obesity on Perivascular Adipose Tissue Vasorelaxant Function: Nitric Oxide, Inflammation and Elevated Systemic Blood Pressure

Journal of Vascular Research

Volumn 52, Issue 5, 2016, Pages 299-305

  Aghamohammadzadeh, Reza ^a   Unwin, Richard D ^a,b   Greenstein, Adam S ^a   Heagerty, Anthony M ^a  

^a UNIVERSITY OF MANCHESTER   (United Kingdom)

^b MANCHESTER UNIVERSITY NHS FOUNDATION TRUST   (United Kingdom)

Author keywords

Contractility; Endothelium; Nitric oxide; Obesity; Perivascular adipose tissue; Superoxide dismutase

Indexed keywords

ADIPONECTIN; C REACTIVE PROTEIN; CATALASE; GLUCOSE; HIGHLY SENSITIVE C REACTIVE PROTEIN; INSULIN; INTERLEUKIN 6; LEPTIN; NITRIC OXIDE; PEROXIREDOXIN 1; RESISTIN; SUPEROXIDE DISMUTASE; TRIACYLGLYCEROL; TUMOR NECROSIS FACTOR ALPHA; UNCLASSIFIED DRUG; COPPER ZINC SUPEROXIDE DISMUTASE; ENZYME INHIBITOR; NITRIC OXIDE SYNTHASE; PEROXIREDOXIN; PRDX1 PROTEIN, HUMAN; SOD1 PROTEIN, HUMAN; SOD1 PROTEIN, RAT; VASOCONSTRICTOR AGENT;

ADIPOSE TISSUE; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CLINICAL ARTICLE; CONTROLLED STUDY; ELEVATED BLOOD PRESSURE; HUMAN; INFLAMMATION; LIPID DIET; MALE; MESENTERY BLOOD VESSEL; MYOGRAPHY; NONHUMAN; OBESITY; PERIVASCULAR ADIPOSE TISSUE; PRIORITY JOURNAL; RAT; SYSTOLIC BLOOD PRESSURE; VASODILATATION; ANIMAL; ANTAGONISTS AND INHIBITORS; BLOOD PRESSURE; CASE CONTROL STUDY; DISEASE MODEL; DOSE RESPONSE; DRUG EFFECTS; MESENTERIC ARTERY; METABOLISM; MIDDLE AGED; PARACRINE SIGNALING; PATHOPHYSIOLOGY; PROTEOMICS; SIGNAL TRANSDUCTION; SPRAGUE DAWLEY RAT; VASOCONSTRICTION;

ADIPOSE TISSUE; ANIMALS; BLOOD PRESSURE; CASE-CONTROL STUDIES; DISEASE MODELS, ANIMAL; DOSE-RESPONSE RELATIONSHIP, DRUG; ENZYME INHIBITORS; HUMANS; INFLAMMATION; MALE; MESENTERIC ARTERIES; MIDDLE AGED; NITRIC OXIDE; NITRIC OXIDE SYNTHASE; OBESITY; PARACRINE COMMUNICATION; PEROXIREDOXINS; PROTEOMICS; RATS, SPRAGUE-DAWLEY; SIGNAL TRANSDUCTION; SUPEROXIDE DISMUTASE; SUPEROXIDE DISMUTASE-1; VASOCONSTRICTION; VASOCONSTRICTOR AGENTS; VASODILATION;

EID: 84959255056     PISSN: 10181172     EISSN: 14230135     Source Type: Journal    
DOI: 10.1159/000443885     Document Type: Article

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