Data supporting the activation of autophagy genes in the diabetic heart

Data in Brief

Volumn 5, Issue , 2015, Pages 269-275

  Munasinghe, Pujika Emani ^a   Riu, Federica ^b,c   Dixit, Parul ^a   Edamatsu, Midori ^a,d   Saxena, Pankaj ^a   Hamer, Nathan S J ^a   Galvin, Ivor F ^a   Bunton, Richard W ^a   Lequeux, Sharon ^a   Jones, Greg ^a   Lamberts, Regis R ^a   Emanueli, Costanza ^b   Madeddu, Paolo ^b   Katare, Rajesh ^a  

^a UNIVERSITY OF OTAGO   (New Zealand)

^b UNIVERSITY OF BRISTOL   (United Kingdom)

^c UNIVERSITY OF NOTTINGHAM   (United Kingdom)

^d OKAYAMA UNIVERSITY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

AUTOPHAGY; AUTOPHAGY GENES; CULTURED CARDIOMYOCYTES; HIGH GLUCOSE; HUMAN HEART; IN-VITRO; TYPE-2 DIABETES;

GENES;

EID: 84942249744     PISSN: None     EISSN: 23523409     Source Type: Journal    
DOI: 10.1016/j.dib.2015.09.003     Document Type: DP

References (8)

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2. Katare R., Caporali A., Zentilin L., Avolio E., Sala-Newby G., Oikawa A., et al. Intravenous gene therapy with PIM-1 via a cardiotropic viral vector halts the progression of diabetic cardiomyopathy through promotion of prosurvival signaling. Circ. Res. 2011, 108:1238-1251.
3. Katare R.G., Caporali A., Oikawa A., Meloni M., Emanueli C., Madeddu P. Vitamin B1 analog benfotiamine prevents diabetes-induced diastolic dysfunction and heart failure through Akt/Pim-1-mediated survival pathway. Circ. Heart Fail. 2010, 3:294-305.
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6. King N., McGivan J.D., Griffiths E.J., Halestrap A.P., Suleiman M.S. Glutamate loading protects freshly isolated and perfused adult cardiomyocytes against intracellular ROS generation. J. Mol. Cell Cardiol. 2003, 35:975-984.
7. Moore A., Shindikar A., Fomison-Nurse I., Riu F., Munasinghe P.E., Ram T.P., et al. Rapid onset of cardiomyopathy in STZ-induced female diabetic mice involves the downregulation of pro-survival Pim-1. Cardiovasc. Diabetol. 2014, 13:68.
8. Katare R., Oikawa A., Cesselli D., Beltrami A.P., Avolio E., Muthukrishnan D., et al. Boosting the pentose phosphate pathway restores cardiac progenitor cell availability in diabetes. Cardiovasc. ReS. 2013, 97:55-65.