Omics-based depression and inflammation research

Revista Brasileira de Psiquiatria

Volumn 37, Issue 1, 2015, Pages 1-2

  Maes, Michael ^a,b,c   Noto, Cristiano ^d   Brietzke, Elisa ^d  

^a CHULALONGKORN UNIVERSITY   (Thailand)

^b DEAKIN UNIVERSITY   (Australia)

^c STATE UNIVERSITY OF LONDRINA   (Brazil)

^d FEDERAL UNIVERSITY OF SÃO PAULO   (Brazil)

Author keywords

[No Author keywords available]

Indexed keywords

DEPRESSION; EDITORIAL; EPIGENETICS; GENOMICS; HUMAN; INFLAMMATION; MEDICAL RESEARCH; METABOLOMICS; MOLECULAR BIOLOGY; PROTEOMICS; TRANSCRIPTOMICS; BIPOLAR DISORDER; FEMALE; IMMUNOLOGY; MALE; METABOLISM; OXIDATIVE STRESS; PHYSIOLOGY; TRENDS;

REACTIVE NITROGEN SPECIES; SIGNAL PEPTIDE;

BIOMEDICAL RESEARCH; BIPOLAR DISORDER; DEPRESSIVE DISORDER; FEMALE; HUMANS; INFLAMMATION; INTRACELLULAR SIGNALING PEPTIDES AND PROTEINS; MALE; OXIDATIVE STRESS; REACTIVE NITROGEN SPECIES;

EID: 84941951492     PISSN: 15164446     EISSN: None     Source Type: Journal    
DOI: 10.1590/1516-4446-2015-3609     Document Type: Editorial

References (5)

1. Leonard B, Maes M. Mechanistic explanations how cell-mediated immune activation, inflammation and oxidative and nitrosative stress pathways and their sequels and concomitants play a role in the pathophysiology of unipolar depression. Neurosci Biobehav Rev. 2012;36:764-85.
2. Moylan S, Maes M, Wray NR, Berk M. The neuroprogressive nature of major depressive disorder: pathways to disease evolution and resistance, and therapeutic implications. Mol Psychiatry. 2013; 18:595-606.
3. Maes M, Kubera M, Obuchowiczwa E, Goehler L, Brzeszcz J. Depression’s multiple comorbidities explained by (neuro)inflammatory and oxidative & nitrosative stress pathways. Neuro Endocrinol Lett. 2011;32:7-24.
4. Kubera M, Obuchowicz E, Goehler L, Brzeszcz J, Maes M. In animal models, psychosocial stress-induced (neuro)inflammation, apoptosis and reduced neurogenesis are associated to the onset of depression. Prog Neuropsychopharmacol Biol Psychiatry. 2011;35:744-59.
5. Maes M, Fisar Z, Medina M, Scapagnini G, Nowak G, Berk M. New drug targets in depression: inflammatory, cell-mediated immune, oxidative and nitrosative stress,mitochondrial, antioxidant, and neuroprogressive pathways. And new drug candidates–Nrf2 activators and GSK-3 inhibitors. Inflammopharmacology. 2012; 20:127-50.