N-Acetyl Cysteine improves the diabetic cardiac function: Possible role of fibrosis inhibition

BMC Cardiovascular Disorders

Volumn 15, Issue 1, 2015, Pages

  Liu, Cong ^a   Lu, Xiao Zhao ^b   Shen, Ming Zhi ^b   Xing, Chang Yang ^a   Ma, Jing ^a   Duan, Yun You ^a   Yuan, Li Jun ^a  

^a FOURTH MILITARY MEDICAL UNIVERSITY   (China)

^b FOURTH MILITARY MEDICAL UNIVERSITY   (China)

Author keywords

Cardiac function; Diabetic cardiomyopathy; Fibrosis; N acetyl L cysteine; Reactive oxygen species

Indexed keywords

ACETYLCYSTEINE; CHOLECYSTOKININ OCTAPEPTIDE; COLLAGEN; GLUCOSE;

ANIMAL CELL; ANIMAL CELL CULTURE; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL ISOLATION; CELL PROLIFERATION; COLLAGEN SYNTHESIS; CONTROLLED STUDY; DIABETIC CARDIOMYOPATHY; DIASTOLE; DIET RESTRICTION; DRUG MECHANISM; ECHOCARDIOGRAPHY; FIBROBLAST; GENE EXPRESSION; HEART; HEART FUNCTION; HEART MUSCLE FIBROSIS; HEART PROTECTION; HEART VENTRICLE REMODELING; HYPERGLYCEMIA; MALE; MOUSE; NONHUMAN; POLYMERASE CHAIN REACTION; PRIORITY JOURNAL; STREPTOZOTOCIN-INDUCED DIABETES MELLITUS; STRESS; SYSTOLE; ANIMAL; C57BL MOUSE; CELL CULTURE; DIABETES MELLITUS, EXPERIMENTAL; DIABETIC CARDIOMYOPATHIES; DRUG EFFECTS; FIBROSIS; METABOLISM; OXIDATIVE STRESS; PATHOLOGY; PHYSIOLOGY;

ACETYLCYSTEINE; ANIMALS; CELLS, CULTURED; DIABETES MELLITUS, EXPERIMENTAL; DIABETIC CARDIOMYOPATHIES; FIBROSIS; MALE; MICE; MICE, INBRED C57BL; OXIDATIVE STRESS;

EID: 84938605039     PISSN: None     EISSN: 14712261     Source Type: Journal    
DOI: 10.1186/s12872-015-0076-3     Document Type: Article

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