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Volumn 54, Issue 4, 2014, Pages 747-748

Abnormalities of selenium but not of copperhomeostasis may drive tissue fibrosis in patients with systemic sclerosis

Author keywords

[No Author keywords available]

Indexed keywords

ALBUMIN; BIOCHEMICAL MARKER; CERULOPLASMIN; COPPER; HEMOGLOBIN A1C; SELENIUM; ZINC;

EID: 84926510261     PISSN: 14620324     EISSN: 14620332     Source Type: Journal    
DOI: 10.1093/rheumatology/keu470     Document Type: Letter
Times cited : (8)

References (8)
  • 2
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    • Demonstration of a hyperglycemia-driven pathogenic abnormality of copper homeostasis in diabetes and its reversibility by selective chelation: quantitative comparisons between the biology of copper and eight other nutritionally essential elements in normal and diabetic individuals
    • Cooper GJS, Chan YK, Dissanayake AM et al. Demonstration of a hyperglycemia-driven pathogenic abnormality of copper homeostasis in diabetes and its reversibility by selective chelation: quantitative comparisons between the biology of copper and eight other nutritionally essential elements in normal and diabetic individuals. Diabetes 2005;54:1468-76.
    • (2005) Diabetes , vol.54 , pp. 1468-1476
    • Cooper, G.J.S.1    Chan, Y.K.2    Dissanayake, A.M.3
  • 3
    • 84861914251 scopus 로고    scopus 로고
    • Selective divalent copper chelation for the treatment of diabetes mellitus
    • Cooper GJ. Selective divalent copper chelation for the treatment of diabetes mellitus. Curr Med Chem 2012;19:2828-60.
    • (2012) Curr Med Chem , vol.19 , pp. 2828-2860
    • Cooper, G.J.1
  • 4
    • 49249098236 scopus 로고    scopus 로고
    • A copper (II)-selective chelator ameliorates diabetes-evoked renal fibrosis and albuminuria, and suppresses pathogenic TGF-beta activation in the kidneys of rats used as a model of diabetes
    • Gong D, Lu J, Chen X et al. A copper (II)-selective chelator ameliorates diabetes-evoked renal fibrosis and albuminuria, and suppresses pathogenic TGF-beta activation in the kidneys of rats used as a model of diabetes. Diabetologia 2008;51:1741-51.
    • (2008) Diabetologia , vol.51 , pp. 1741-1751
    • Gong, D.1    Lu, J.2    Chen, X.3
  • 5
    • 0017200150 scopus 로고
    • Serum copper and caeruloplasmin in ankylosing spondylitis, systemic sclerosis, and morphea
    • Jayson MI, Davis P, Whicher JT, Walters G. Serum copper and caeruloplasmin in ankylosing spondylitis, systemic sclerosis, and morphea. Ann Rheum Dis 1975;35:443-5.
    • (1975) Ann Rheum Dis , vol.35 , pp. 443-445
    • Jayson, M.I.1    Davis, P.2    Whicher, J.T.3    Walters, G.4
  • 6
    • 0026782862 scopus 로고
    • Dietary intake and nutritional status in patients with systemic sclerosis
    • Lundberg AC, Akesson A, Akesson B. Dietary intake and nutritional status in patients with systemic sclerosis. Ann Rheum Dis 1992;51:1143-8.
    • (1992) Ann Rheum Dis , vol.51 , pp. 1143-1148
    • Lundberg, A.C.1    Akesson, A.2    Akesson, B.3
  • 7
    • 0028026729 scopus 로고
    • Micronutrient antioxidant status in patients with primary Raynaud's phenomenon and systemic sclerosis
    • Herrick AL, Rieley F, Schofield D et al. Micronutrient antioxidant status in patients with primary Raynaud's phenomenon and systemic sclerosis. J Rheumatol 1994;21:1477-83.
    • (1994) J Rheumatol , vol.21 , pp. 1477-1483
    • Herrick, A.L.1    Rieley, F.2    Schofield, D.3
  • 8
    • 0009680686 scopus 로고    scopus 로고
    • Selenium deficiency and thyroid fibrosis A key role for macrophages and transforming growth factor beta (TGF-b).
    • Contempre B, Le Moine O, Dumont JE et al. Selenium deficiency and thyroid fibrosis. A key role for macrophages and transforming growth factor beta (TGF-b). Mol Cell Endocrinol 1996;124:7-15.
    • (1996) Mol Cell Endocrinol , vol.124 , pp. 7-15
    • Contempre, B.1    Le Moine, O.2    Dumont, J.E.3


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.