Mitochondrial N-formyl peptides induce cardiovascular collapse and sepsis-like syndrome

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 308, Issue 7, 2015, Pages 768-777

  Wenceslau, Camilla Ferreira ^a   McCarthy, Cameron G ^a   Szasz, Theodora ^a   Goulopoulou, Styliani ^a   Clinton Webb, R ^a  

^a MEDICAL COLLEGE OF GEORGIA   (United States)

Author keywords

Cardiovascular collapse; Mitochondrial N formyl peptides; Sepsis like syndrome

Indexed keywords

ANTIHISTAMINIC AGENT; FORMYLPEPTIDE RECEPTOR; MITOCHONDRIAL DNA; MITOCHONDRIAL N FORMYL PEPTIDE; MITOCHONDRIAL PROTEIN; NITRIC OXIDE; REACTIVE OXYGEN METABOLITE; UNCLASSIFIED DRUG; MITOGEN ACTIVATED PROTEIN KINASE; N-FORMYLMETHIONYL-METHIONYL-TYROSYL-ALANYL-LEUCYL-PHENYLALANINE; OLIGOPEPTIDE;

AMINO ACID SEQUENCE; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BLOOD PRESSURE; BLOOD VESSEL PERMEABILITY; CARCINOGENESIS; CARDIOVASCULAR PARAMETERS; CELL AGING; CELL DAMAGE; CELL DEATH; CELL DIFFERENTIATION; CONCENTRATION RESPONSE; CONTROLLED STUDY; HISTAMINE RELEASE; HYPOTENSION; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROTEIN BLOOD LEVEL; PROTEIN EXPRESSION; RAT; SHOCK; SYSTEMIC INFLAMMATORY RESPONSE SYNDROME; AGONISTS; ANIMAL; BASOPHIL; BLOOD CLOTTING; CAPILLARY PERMEABILITY; CHEMICALLY INDUCED; DOSE RESPONSE; DRUG EFFECTS; FEVER; LUNG INJURY; METABOLISM; PATHOPHYSIOLOGY; SEPSIS; SIGNAL TRANSDUCTION; TIME; VASODILATATION; WISTAR RAT;

ANIMALS; BASOPHILS; BLOOD COAGULATION; CAPILLARY PERMEABILITY; DOSE-RESPONSE RELATIONSHIP, DRUG; FEVER; HISTAMINE RELEASE; HYPOTENSION; LUNG INJURY; MITOCHONDRIAL PROTEINS; MITOGEN-ACTIVATED PROTEIN KINASES; NITRIC OXIDE; OLIGOPEPTIDES; RATS, WISTAR; RECEPTORS, FORMYL PEPTIDE; SEPSIS; SHOCK; SIGNAL TRANSDUCTION; TIME FACTORS; VASODILATION;

EID: 84926318798     PISSN: 03636135     EISSN: 15221539     Source Type: Journal    
DOI: 10.1152/ajpheart.00779.2014     Document Type: Article

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