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Volumn 64, Issue 6, 2014, Pages 1192-1193

Endothelial dysfunction potentially interacts with impaired glucose metabolism to increase cardiovascular risk

Author keywords

Cardiovascular disease; Population; Type 2 diabetes mellitus

Indexed keywords

ALDOSTERONE; ANGIOTENSIN II; ARGININE; CALCIUM; DIPEPTIDYL CARBOXYPEPTIDASE; ENDOTHELIAL LEUKOCYTE ADHESION MOLECULE 1; ENDOTHELIN 1; INSULIN; INSULIN RECEPTOR SUBSTRATE; INTERCELLULAR ADHESION MOLECULE 1; METFORMIN; MITOGEN ACTIVATED PROTEIN KINASE; NITRIC OXIDE; NITRIC OXIDE SYNTHASE; PHOSPHATIDYLINOSITIDE 3 KINASE; PHOSPHOTRANSFERASE; PROSTACYCLIN; PROTEIN KINASE B; RAMIPRIL; ROSIGLITAZONE; S6 KINASE; THROMBOXANE A2; UNCLASSIFIED DRUG; VON WILLEBRAND FACTOR;

EID: 84922481654     PISSN: 0194911X     EISSN: 15244563     Source Type: Journal    
DOI: 10.1161/HYPERTENSIONAHA.114.04348     Document Type: Note
Times cited : (32)

References (10)
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    • Activation of mTOR/p70S6 kinase by ANG II inhibits insulin-stimulated endothelial nitric oxide synthase and vasodilation
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  • 7
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    • Endothelial dysfunction and low-grade infammation explain much of the excess cardiovascular mortality in individuals with type 2 diabetes: The Hoorn Study
    • De Jager J, Dekker JM, Kooy A, Kostense PJ, Nijpels G, Heine RJ, Bouter LM, Stehouwer CD. Endothelial dysfunction and low-grade infammation explain much of the excess cardiovascular mortality in individuals with type 2 diabetes: the Hoorn Study. Arterioscler Thromb Vasc Biol. 2006;26:1086-1093.
    • (2006) Arterioscler Thromb Vasc Biol. , vol.26 , pp. 1086-1093
    • De Jager, J.1    Dekker, J.M.2    Kooy, A.3    Kostense, P.J.4    Nijpels, G.5    Heine, R.J.6    Bouter, L.M.7    Stehouwer, C.D.8
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.