Dissociation of mitochondrial from sarcoplasmic reticular stress in Drosophila cardiomyopathy induced by molecularly distinct mitochondrial fusion defects

Journal of Molecular and Cellular Cardiology

Volumn 80, Issue , 2015, Pages 71-80

  Bhandari, Poonam ^a   Song, Moshi ^a   Dorn, Gerald W ^a  

^a WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Drosophila; Endoplasmic reticular stress; Mitochondrial fusion; Mitofusin; Optic atrophy 1; Reactive oxygen species

Indexed keywords

CELL PROTEIN; DROSOPHILA PROTEIN; MITOFUSIN; PROTEIN DRP1; PROTEIN OPA1; PROTEIN ROMO1; REACTIVE OXYGEN METABOLITE; SUPEROXIDE DISMUTASE; UNCLASSIFIED DRUG; X BOX BINDING PROTEIN 1; COPPER ZINC SUPEROXIDE DISMUTASE; CYTOSKELETON PROTEIN; DRP1 PROTEIN, DROSOPHILA; GUANINE NUCLEOTIDE BINDING PROTEIN; MANGANESE SUPEROXIDE DISMUTASE; MEMBRANE PROTEIN; MFN2 PROTEIN, DROSOPHILA; OPA1 PROTEIN, DROSOPHILA;

ARTICLE; CARDIOMYOPATHY; CONTROLLED STUDY; DISEASE COURSE; DISORDERS OF MITOCHONDRIAL FUNCTIONS; DROSOPHILA; ENDOPLASMIC RETICULUM STRESS; ENZYME INHIBITION; HEART DISEASE; HEART FUNCTION; HEART MITOCHONDRION; HEART MUSCLE CELL; MEMBRANE DEPOLARIZATION; MITOCHONDRIAL FUSION DEFECT; MITOCHONDRIAL MEMBRANE POTENTIAL; MITOCHONDRIAL VOLUME; MOLECULAR BIOLOGY; NONHUMAN; PATHOPHYSIOLOGY; PROTEIN DEPLETION; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN LOCALIZATION; RNA INTERFERENCE; SARCOPLASMIC RETICULUM; ANIMAL; ANTIBODY SPECIFICITY; DEFICIENCY; DISEASE MODEL; FEMALE; GENE EXPRESSION; GENE SILENCING; GENETICS; MALE; METABOLISM; MITOCHONDRIAL DYNAMICS; MITOCHONDRION; PATHOLOGY; TRANSGENIC ANIMAL;

ANIMALS; ANIMALS, GENETICALLY MODIFIED; CARDIOMYOPATHIES; CYTOSKELETAL PROTEINS; DISEASE MODELS, ANIMAL; DROSOPHILA; DROSOPHILA PROTEINS; ENDOPLASMIC RETICULUM STRESS; FEMALE; GENE EXPRESSION; GENE KNOCKDOWN TECHNIQUES; GTP-BINDING PROTEINS; MALE; MEMBRANE PROTEINS; MITOCHONDRIA; MITOCHONDRIAL DYNAMICS; MYOCYTES, CARDIAC; ORGAN SPECIFICITY; REACTIVE OXYGEN SPECIES; SARCOPLASMIC RETICULUM; SUPEROXIDE DISMUTASE;

EID: 84920871175     PISSN: 00222828     EISSN: 10958584     Source Type: Journal    
DOI: 10.1016/j.yjmcc.2014.12.018     Document Type: Article

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