-
1
-
-
4344692876
-
Spectrum of acute renal failure in the intensive care unit: The PICARD experience
-
Mehta RL, Pascual MT, Soroko S, Savage BR, Himmelfarb J, et al. (2004) Spectrum of acute renal failure in the intensive care unit: the PICARD experience. Kidney Int 66(4): 1613-1621.
-
(2004)
Kidney Int
, vol.66
, Issue.4
, pp. 1613-1621
-
-
Mehta, R.L.1
Pascual, M.T.2
Soroko, S.3
Savage, B.R.4
Himmelfarb, J.5
-
2
-
-
84891505671
-
Ischemia-reperfusion induces renal tubule pyroptosis via the chop-caspase-11 pathway
-
Yang JR, Yao FH, Zhang JG, Ji ZY, Li KL, et al. (2014) Ischemia-reperfusion induces renal tubule pyroptosis via the CHOP-caspase-11 pathway. Am J Physiol Renal Physiol 306(1): F75-84.
-
(2014)
Am J Physiol Renal Physiol
, vol.306
, Issue.1
, pp. F75-F84
-
-
Yang, J.R.1
Yao, F.H.2
Zhang, J.G.3
Ji, Z.Y.4
Li, K.L.5
-
3
-
-
33749492425
-
Endoplasmic reticulum stress signaling in disease
-
Marciniak SJ, Ron D (2006) Endoplasmic reticulum stress signaling in disease. Physiol Rev 86(4): 1133-1149.
-
(2006)
Physiol Rev
, vol.86
, Issue.4
, pp. 1133-1149
-
-
Marciniak, S.J.1
Ron, D.2
-
4
-
-
84864695256
-
Endoplasmic reticulum stress modulates liver inflammatory immune response in the pathogenesis of liver ischemia and reperfusion injury
-
Liu J, Ren F, Cheng Q, Bai L, Shen X, et al. (2012) Endoplasmic reticulum stress modulates liver inflammatory immune response in the pathogenesis of liver ischemia and reperfusion injury. Transplantation 94(3): 211-217.
-
(2012)
Transplantation
, vol.94
, Issue.3
, pp. 211-217
-
-
Liu, J.1
Ren, F.2
Cheng, Q.3
Bai, L.4
Shen, X.5
-
5
-
-
84869785004
-
Atypical activation of the unfolded protein response in cystic fibrosis airway cells contributes to p38 mapk-mediated innate immune responses
-
Blohmke CJ, Mayer ML, Tang AC, Hirschfeld AF, Fjell CD, et al. (2012) Atypical activation of the unfolded protein response in cystic fibrosis airway cells contributes to p38 MAPK-mediated innate immune responses. J Immunol 189(11): 5467-5475.
-
(2012)
J Immunol
, vol.189
, Issue.11
, pp. 5467-5475
-
-
Blohmke, C.J.1
Mayer, M.L.2
Tang, A.C.3
Hirschfeld, A.F.4
Fjell, C.D.5
-
6
-
-
1842854719
-
Ischemiainduced neuronal cell death is mediated by the endoplasmic reticulum stress pathway involving chop
-
Tajiri S, Oyadomari S, Yano S, Morioka M, Gotoh T, et al. (2004) Ischemiainduced neuronal cell death is mediated by the endoplasmic reticulum stress pathway involving CHOP. Cell Death Differ 11(4): 403-415.
-
(2004)
Cell Death Differ
, vol.11
, Issue.4
, pp. 403-415
-
-
Tajiri, S.1
Oyadomari, S.2
Yano, S.3
Morioka, M.4
Gotoh, T.5
-
7
-
-
79955597257
-
C/ebp homologous protein deficiency attenuates myocardial reperfusion injury by inhibiting myocardial apoptosis and inflammation
-
Miyazaki Y, Kaikita K, Endo M, Horio E, Miura M, et al. (2011) C/EBP homologous protein deficiency attenuates myocardial reperfusion injury by inhibiting myocardial apoptosis and inflammation. Arterioscler Thromb Vasc Biol 31(5): 1124-1132.
-
(2011)
Arterioscler Thromb Vasc Biol
, vol.31
, Issue.5
, pp. 1124-1132
-
-
Miyazaki, Y.1
Kaikita, K.2
Endo, M.3
Horio, E.4
Miura, M.5
-
8
-
-
84893322248
-
Lipopolysaccharide-induced cross-tolerance against renal ischemia-reperfusion injury is mediated by hypoxiainducible factor-2alpha-regulated nitric oxide production
-
He K, Chen X, Han C, Xu L, Zhang J, et al. (2014) Lipopolysaccharide-induced cross-tolerance against renal ischemia-reperfusion injury is mediated by hypoxiainducible factor-2alpha-regulated nitric oxide production. Kidney Int 85(2): 276-288.
-
(2014)
Kidney Int
, vol.85
, Issue.2
, pp. 276-288
-
-
He, K.1
Chen, X.2
Han, C.3
Xu, L.4
Zhang, J.5
-
9
-
-
84856276113
-
Transient ureteral obstruction prevents against kidney ischemia/reperfusion injury via hypoxiainducible factor (HIF)-2alpha activation
-
Zhang S, Han CH, Chen XS, Zhang M, Xu LM, et al. (2012) Transient ureteral obstruction prevents against kidney ischemia/reperfusion injury via hypoxiainducible factor (HIF)-2alpha activation. PLoS One 7(1): e29876.
-
(2012)
PLoS One
, vol.7
, Issue.1
, pp. e29876
-
-
Zhang, S.1
Han, C.H.2
Chen, X.S.3
Zhang, M.4
Xu, L.M.5
-
10
-
-
84864008957
-
C/ebp homologous protein (CHOP) deficiency aggravates hippocampal cell apoptosis and impairs memory performance
-
Chen CM, Wu CT, Chiang CK, Liao BW, Liu SH (2012) C/EBP homologous protein (CHOP) deficiency aggravates hippocampal cell apoptosis and impairs memory performance. PLoS One 7(7): e40801.
-
(2012)
PLoS One
, vol.7
, Issue.7
, pp. e40801
-
-
Chen, C.M.1
Wu, C.T.2
Chiang, C.K.3
Liao, B.W.4
Liu, S.H.5
-
11
-
-
84874322294
-
CHOP regulates the p53-mdm2 axis and is required for neuronal survival after seizures
-
Engel T, Sanz-Rodgriguez A, Jimenez-Mateos EM, Concannon CG, Jimenez- Pacheco A, et al. (2013) CHOP regulates the p53-MDM2 axis and is required for neuronal survival after seizures. Brain 136(Pt 2): 577-592.
-
(2013)
Brain
, vol.136
, Issue.2
, pp. 577-592
-
-
Engel, T.1
Sanz-Rodgriguez, A.2
Jimenez-Mateos, E.M.3
Concannon, C.G.4
Jimenez- Pacheco, A.5
-
12
-
-
77954377779
-
The endoplasmic reticulum stress response factor chop-10 protects against hypoxia-induced neuronal death
-
Halterman MW, Gill M, Dejesus C, Ogihara M, Schor NF, et al. (2010) The endoplasmic reticulum stress response factor CHOP-10 protects against hypoxia-induced neuronal death. J Biol Chem 285(28): 21329-21340.
-
(2010)
J Biol Chem
, vol.285
, Issue.28
, pp. 21329-21340
-
-
Halterman, M.W.1
Gill, M.2
Dejesus, C.3
Ogihara, M.4
Schor, N.F.5
-
13
-
-
79954986952
-
Pkr-dependent CHOP induction limits hyperoxia-induced lung injury
-
Lozon TI, Eastman AJ, Matute-Bello G, Chen P, Hallstrand TS, et al. (2011) PKR-dependent CHOP induction limits hyperoxia-induced lung injury. Am J Physiol Lung Cell Mol Physiol 300(3): L422-429.
-
(2011)
Am J Physiol Lung Cell Mol Physiol
, vol.300
, Issue.3
, pp. L422-L429
-
-
Lozon, T.I.1
Eastman, A.J.2
Matute-Bello, G.3
Chen, P.4
Hallstrand, T.S.5
-
14
-
-
84874077537
-
CHOP deficiency results in elevated lipopolysaccharide-induced inflammation and kidney injury
-
Esposito V, Grosjean F, Tan J, Huang L, Zhu L, et al. (2013) CHOP deficiency results in elevated lipopolysaccharide-induced inflammation and kidney injury. Am J Physiol Renal Physiol 304(4): F440-450.
-
(2013)
Am J Physiol Renal Physiol
, vol.304
, Issue.4
, pp. F440-F450
-
-
Esposito, V.1
Grosjean, F.2
Tan, J.3
Huang, L.4
Zhu, L.5
-
15
-
-
84892393658
-
The stress-regulated transcription factor CHOP promotes hepatic inflammatory gene expression, fibrosis, and oncogenesis
-
DeZwaan-McCabe D, Riordan JD, Arensdorf AM, Icardi MS, Dupuy AJ, et al. (2013) The stress-regulated transcription factor CHOP promotes hepatic inflammatory gene expression, fibrosis, and oncogenesis. PLoS Genet 9(12): e1003937.
-
(2013)
PLoS Genet
, vol.9
, Issue.12
, pp. e1003937
-
-
DeZwaan-McCabe, D.1
Riordan, J.D.2
Arensdorf, A.M.3
Icardi, M.S.4
Dupuy, A.J.5
-
16
-
-
84879568119
-
C/ebp homologous protein-induced macrophage apoptosis protects mice from steatohepatitis
-
Malhi H, Kropp EM, Clavo V, Kobrossi CR, Han J, et al. (2013) C/EBP homologous protein-induced macrophage apoptosis protects mice from steatohepatitis. J Biol Chem 288(26): 18624-18642.
-
(2013)
J Biol Chem
, vol.288
, Issue.26
, pp. 18624-18642
-
-
Malhi, H.1
Kropp, E.M.2
Clavo, V.3
Kobrossi, C.R.4
Han, J.5
-
17
-
-
0024238881
-
Mediators of ischemic renal injury
-
Bonventre JV. (1988) Mediators of ischemic renal injury. Annu Rev Med 39: 531-544.
-
(1988)
Annu Rev Med
, vol.39
, pp. 531-544
-
-
Bonventre, J.V.1
-
18
-
-
33846912184
-
Vascular abnormalities in mice deficient for the G protein-coupled receptor gpr4 that functions as a ph sensor
-
Yang LV, Radu CG, Roy M, Lee S, Mclaughlin J, et al. (2007) Vascular abnormalities in mice deficient for the G protein-coupled receptor GPR4 that functions as a pH sensor. Mol Cell Biol 27(4): 1334-1347.
-
(2007)
Mol Cell Biol
, vol.27
, Issue.4
, pp. 1334-1347
-
-
Yang, L.V.1
Radu, C.G.2
Roy, M.3
Lee, S.4
Mclaughlin, J.5
-
19
-
-
77957869783
-
Deletion of the ph sensor gpr4 decreases renal acid excretion
-
Sun X, Yang LV, Tiegs BC, Arend LJ, McGraw DW, et al. (2010) Deletion of the pH sensor GPR4 decreases renal acid excretion. J Am Soc Nephrol 21(10): 1745-1755.
-
(2010)
J Am Soc Nephrol
, vol.21
, Issue.10
, pp. 1745-1755
-
-
Sun, X.1
Yang, L.V.2
Tiegs, B.C.3
Arend, L.J.4
McGraw, D.W.5
-
20
-
-
81155128295
-
Activation of gpr4 by acidosis increases endothelial cell adhesion through the camp/epac pathway
-
Chen A, Dong L, Leffler NR, Asch AS, Witte ON, et al. (2011) Activation of GPR4 by acidosis increases endothelial cell adhesion through the cAMP/Epac pathway. PLoS One 6(11): e27586.
-
(2011)
PLoS One
, vol.6
, Issue.11
, pp. e27586
-
-
Chen, A.1
Dong, L.2
Leffler, N.R.3
Asch, A.S.4
Witte, O.N.5
-
21
-
-
84876181793
-
Acidosis activation of the proton-sensing gpr4 receptor stimulates vascular endothelial cell inflammatory responses revealed by transcriptome analysis
-
Dong L, Li Z, Leffler NR, Asch AS, Chi JT, et al. (2013) Acidosis activation of the proton-sensing GPR4 receptor stimulates vascular endothelial cell inflammatory responses revealed by transcriptome analysis. PLoS One 8(4): e61991.
-
(2013)
PLoS One
, vol.8
, Issue.4
, pp. e61991
-
-
Dong, L.1
Li, Z.2
Leffler, N.R.3
Asch, A.S.4
Chi, J.T.5
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