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Volumn 22, Issue 8, 2014, Pages 428-429

Porphyromonas gingivalis neutrophil manipulation: Risk factor for periodontitis?

Author keywords

[No Author keywords available]

Indexed keywords

CHEMOKINE; COMPLEMENT COMPONENT C5A RECEPTOR; LIPID A; PHOSPHATIDYLINOSITOL 3 KINASE; TOLL LIKE RECEPTOR 2; TOLL LIKE RECEPTOR 4;

EID: 84905560545     PISSN: 0966842X     EISSN: 18784380     Source Type: Journal    
DOI: 10.1016/j.tim.2014.06.006     Document Type: Short Survey
Times cited : (11)

References (10)
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  • 3
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    • Porphyromonas gingivalis manipulates complement and TLR signaling to uncouple bacterial clearance from inflammation and promote dysbiosis
    • Maekawa T., et al. Porphyromonas gingivalis manipulates complement and TLR signaling to uncouple bacterial clearance from inflammation and promote dysbiosis. Cell Host Microbe 2014, 15:768-778.
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  • 4
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    • Low-abundance biofilm species orchestrates inflammatory periodontal disease through the commensal microbiota and complement
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    • (2011) Cell Host Microbe , vol.10 , pp. 497-506
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  • 5
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    • Induction of bone loss by pathobiont-mediated Nod1 signaling in the oral cavity
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  • 6
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    • Smoking and the subgingival ecosystem: a pathogen-enriched community
    • Kumar P.S. Smoking and the subgingival ecosystem: a pathogen-enriched community. Future Microbiol. 2012, 7:917-919.
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    • Kumar, P.S.1
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    • Cornforth D.M., Foster K.R. Competition sensing: the social side of bacterial stress responses. Nat. Rev. Microbiol. 2013, 11:285-293.
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  • 8
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    • Social evolution theory for microorganisms
    • West S.A., et al. Social evolution theory for microorganisms. Nat. Rev. Microbiol. 2006, 4:597-607.
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  • 9
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.