Shock wave induces biological renal damage by activating excessive inflammatory responses in rat model

Inflammation

Volumn 37, Issue 4, 2014, Pages 1317-1325

  Li, Xiang ^a   Long, Qingzhi ^a   Cheng, Xinfa ^a   He, Dalin ^a  

^a XI'AN JIAOTONG UNIVERSITY   (China)

Author keywords

excessive inflammatory responses; inflammatory renal damage; shock wave

Indexed keywords

BETA 2 MICROGLOBULIN; CREATININE; INTERCELLULAR ADHESION MOLECULE 1; INTERLEUKIN 18; INTERLEUKIN 6; MALONALDEHYDE; MONOCYTE CHEMOTACTIC PROTEIN 1; PYRROLIDINE DITHIOCARBAMATE; TUMOR NECROSIS FACTOR ALPHA; CCL2 PROTEIN, RAT; PEROXIDASE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL INFILTRATION; CONTROLLED STUDY; CREATININE BLOOD LEVEL; EXTRACORPOREAL LITHOTRIPSY; GENE EXPRESSION; HISTOLOGY; IMMUNOFLUORESCENCE; IN SITU HYBRIDIZATION; KIDNEY; KIDNEY FIBROSIS; KIDNEY INJURY; KIDNEY TUBULE CELL; MONOCYTE; NEUTROPHIL CHEMOTAXIS; NONHUMAN; OXIDATIVE STRESS; PROTEIN BLOOD LEVEL; PROTEIN EXPRESSION; PROTEIN URINE LEVEL; RAT; UREA NITROGEN BLOOD LEVEL; URINALYSIS; WESTERN BLOTTING; ANIMAL; CYTOLOGY; FIBROSIS; INFLAMMATION; LEUKOCYTE; LITHOTRIPSY; MALE; METABOLISM; NEUTROPHIL; PATHOLOGY; SPRAGUE DAWLEY RAT; URINE;

ANIMALIA; RATTUS;

ANIMALS; CHEMOKINE CCL2; FIBROSIS; INFLAMMATION; INTERCELLULAR ADHESION MOLECULE-1; INTERLEUKIN-18; INTERLEUKIN-6; KIDNEY; LEUKOCYTES; LITHOTRIPSY; MALE; MALONDIALDEHYDE; NEUTROPHILS; PEROXIDASE; RATS; RATS, SPRAGUE-DAWLEY; TUMOR NECROSIS FACTOR-ALPHA;

EID: 84904757514     PISSN: 03603997     EISSN: 15732576     Source Type: Journal    
DOI: 10.1007/s10753-014-9859-4     Document Type: Article

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