High glucose modifies transient receptor potential canonical type 6 channels via increased oxidative stress and syndecan-4 in human podocytes

Biochemical and Biophysical Research Communications

Volumn 450, Issue 1, 2014, Pages 312-317

  Thilo, Florian ^a   Lee, Marlene ^a   Xia, Shengqiang ^b,c   Zakrzewicz, Andreas ^a   Tepel, Martin ^b  

^a CHARITÉ UNIVERSITÄTSMEDIZIN BERLIN   (Germany)

^b ODENSE UNIVERSITY HOSPITAL   (Denmark)

^c TONGJI UNIVERSITY   (China)

Author keywords

Diabetic nephropathy; High glucose; Oxidative stress; Podocytes; Syndecan 4; Transient receptor potential channels

Indexed keywords

CALCIUM; FLUORESCENT DYE; GLUCOSE; REACTIVE OXYGEN METABOLITE; SUPEROXIDE DISMUTASE; SYNDECAN 4; TRANSIENT RECEPTOR POTENTIAL CHANNEL 6;

ARTICLE; CALCIUM TRANSPORT; CONTROLLED STUDY; DOWN REGULATION; GENETIC TRANSFECTION; HUMAN; HUMAN CELL; NUCLEOTIDE SEQUENCE; OXIDATIVE STRESS; PODOCYTE; PRIORITY JOURNAL; PROTEIN EXPRESSION;

DIABETIC NEPHROPATHY; HIGH GLUCOSE; OXIDATIVE STRESS; PODOCYTES; SYNDECAN-4; TRANSIENT RECEPTOR POTENTIAL CHANNELS;

CALCIUM SIGNALING; CELLS, CULTURED; DOSE-RESPONSE RELATIONSHIP, DRUG; GLUCOSE; HUMANS; ION CHANNEL GATING; OXIDATIVE STRESS; OXYGEN; PODOCYTES; SYNDECAN-4; TRPC CATION CHANNELS;

EID: 84904744166     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2014.05.116     Document Type: Article

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