Mutations in VPS26A are not a frequent cause of Parkinson's disease

Neurobiology of Aging

Volumn 35, Issue 6, 2014, Pages 1512.e1-1512.e2

  Koschmidder, Eva ^a   Mollenhauer, Brit ^b,c   Kasten, Meike ^a   Klein, Christine ^a   Lohmann, Katja ^a  

^a UNIVERSITY OF LÜBECK   (Germany)

^b Paracelsus Elena Klinik Kassel   (Germany)

^c UNIVERSITY OF GÖTTINGEN   (Germany)

Author keywords

Parkinson's disease; Retromer complex; VPS26A; VPS35

Indexed keywords

ARTICLE; CONTROLLED STUDY; GENE IDENTIFICATION; GENETIC ASSOCIATION; GENETIC VARIABILITY; HETEROZYGOSITY; HUMAN; MAJOR CLINICAL STUDY; MISSENSE MUTATION; NUCLEOTIDE SEQUENCE; PARKINSON DISEASE; PHENOTYPE; PRIORITY JOURNAL; PROTEIN DOMAIN; PROTEIN SUBUNIT; SINGLE NUCLEOTIDE POLYMORPHISM; ADOLESCENT; ADULT; AGED; CHILD; EPISTASIS; FEMALE; GENETICS; LYSOSOME; MALE; MIDDLE AGED; MITOCHONDRION; MUTATION; SIGNAL TRANSDUCTION; VERY ELDERLY; YOUNG ADULT;

VESICULAR TRANSPORT PROTEIN; VPS26A PROTEIN, HUMAN; VPS35 PROTEIN, HUMAN;

ADOLESCENT; ADULT; AGED; AGED, 80 AND OVER; CHILD; EPISTASIS, GENETIC; FEMALE; GENETIC ASSOCIATION STUDIES; HUMANS; LYSOSOMES; MALE; MIDDLE AGED; MITOCHONDRIA; MUTATION; MUTATION, MISSENSE; PARKINSON DISEASE; SIGNAL TRANSDUCTION; VESICULAR TRANSPORT PROTEINS; YOUNG ADULT;

EID: 84903362808     PISSN: 01974580     EISSN: 15581497     Source Type: Journal    
DOI: 10.1016/j.neurobiolaging.2013.12.016     Document Type: Article

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