Over-expression of catalase in myeloid cells confers acute protection following myocardial infarction

International Journal of Molecular Sciences

Volumn 15, Issue 5, 2014, Pages 9036-9050

  Bernadette Cabigas, E ^a,b   Somasuntharam, Inthirai ^a,b   Brown, Milton E ^a,b   Che, Pao Lin ^a,b   Pendergrass, Karl D ^a,b   Chiang, Bryce ^a   Robert Taylor, W ^a,b   Davis, Michael E ^a,b,c  

^a Emory University   (United States)

^b EMORY UNIVERSITY SCHOOL OF MEDICINE   (United States)

^c Children's Healthcare of Atlanta   (United States)

Author keywords

Antioxidant therapy; Myocardial infarction; Oxidative stress

Indexed keywords

CATALASE; HYDROGEN PEROXIDE; CD45 ANTIGEN; CHEMOKINE; CYTOKINE; PEROXIDASE;

ANGIOGENESIS; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTERY LIGATION; ARTICLE; BONE MARROW CELL; CONTROLLED STUDY; ECHOCARDIOGRAPHY; ENDOTHELIUM CELL; ENZYME ACTIVITY; ENZYME LINKED IMMUNOSORBENT ASSAY; FIBROSIS; FLUORESCENCE ACTIVATED CELL SORTING; GENE OVEREXPRESSION; HEART EJECTION FRACTION; HEART INFARCTION; HEART PROTECTION; INFLAMMATION; ISCHEMIA; MALE; MOUSE; NONHUMAN; OXIDATIVE STRESS; PHENOTYPE; ANIMAL; C57BL MOUSE; CYTOLOGY; DISEASE MODEL; ENZYMOLOGY; METABOLISM; PATHOLOGY; TRANSGENIC MOUSE;

ANIMALS; ANTIGENS, CD45; CATALASE; CHEMOKINES; CYTOKINES; DISEASE MODELS, ANIMAL; ECHOCARDIOGRAPHY; FIBROSIS; HYDROGEN PEROXIDE; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; MYELOID CELLS; MYOCARDIAL INFARCTION; NEOVASCULARIZATION, PHYSIOLOGIC; PEROXIDASES;

EID: 84901351845     PISSN: 16616596     EISSN: 14220067     Source Type: Journal    
DOI: 10.3390/ijms15059036     Document Type: Article

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