Translocation (10;17)(p15;q21): Recurrent anomaly in acute myeloblastic leukemia
Tempescul A, Guillerm G, Douet-Guilbert N, et al. Translocation (10;17)(p15;q21): Recurrent anomaly in acute myeloblastic leukemia. Cancer Genet Cytogenet 2007; 172: 74-76.
Abnormalities of the short arm of chromosome 9 with partial loss of material in hematological disorders
Pollak C, Hagemeijer A. Abnormalities of the short arm of chromosome 9 with partial loss of material in hematological disorders. Leukemia 1987; 1: 541-548.
High EVI1 expression predicts poor survival in acute myeloid leukemia: A study of 319 de novo AML patients
Barjesteh van Waalwijk van Doorn-Khosrovani S, Erpelinck C , van Putten WL, et al. High EVI1 expression predicts poor survival in acute myeloid leukemia: A study of 319 de novo AML patients. Blood 2003; 101: 837-845.
Cytogenetic analysis of 100 consecutive newly diagnosed cases of acute lymphoblastic leukemia in Rio de Janeiro
Silva ML, Ornellas de Souza MH, Ribeiro RC, et al. Cytogenetic analysis of 100 consecutive newly diagnosed cases of acute lymphoblastic leukemia in Rio de Janeiro. Cancer Genet Cytogenet 2002; 137: 85-90.
Using bacterial artificial chromosomes in leukemia research: The experience at the university cytogenetics laboratory in Brest, France
De Braekeleer E, Douet-Guilbert N, Basinko A, et al. Using bacterial artificial chromosomes in leukemia research: The experience at the university cytogenetics laboratory in Brest, France. J Biomed Biotechnol 2011; 2011: 329471.
B S69, anovel adenovirus E1A-Associated protein that inhibits E1A transactivation
Hateboer G, Gennissen A, Ramos Y F, et a l. B S69, anovel adenovirus E1A-Associated protein that inhibits E1A transactivation. EMBO J 1995; 14: 3159-3169.
The adenovirus E1A binding protein BS69 is a corepressor of transcription through recruitment of N-CoR
Masselink H, Bernards R. The adenovirus E1A binding protein BS69 is a corepressor of transcription through recruitment of N-CoR. Oncogene 2000; 19: 1538-1546.
The MYND motif is required for repression of basal transcription from the multidrug resistance 1 promoter by the t(8;21) fusion protein
Lutterbach B, Sun D, Schuetz J, et al. The MYND motif is required for repression of basal transcription from the multidrug resistance 1 promoter by the t(8;21) fusion protein. Mol Cell Biol 1998; 18: 3604-3611.
ETO, fusion partner in t(8;21) acute myeloid leukemia, represses transcription by interaction with the human N-CoR/mSin3/HDAC1 complex
Wang J, Hoshino T, Redner RL, et al. ETO, fusion partner in t(8;21) acute myeloid leukemia, represses transcription by interaction with the human N-CoR/mSin3/HDAC1 complex. Proc Natl Acad Sci USA 1998; 95: 10860-10865.
The conserved Mynd domain of BS69 binds cellular and oncoviral proteins through a common PXLXP motif
Ansieau S, Leutz A. The conserved Mynd domain of BS69 binds cellular and oncoviral proteins through a common PXLXP motif. J Biol Chem 2002; 277: 4906-4910.
The human L(3) MBT polycomb group protein is a transcriptional repressor and interacts physically and functionally with TEL (ETV6)
Boccuni P, MacGrogan D, Scandura JM, et al. The human L(3) MBT polycomb group protein is a transcriptional repressor and interacts physically and functionally with TEL (ETV6). J Biol Chem 2003; 278: 15412-15420.
Methylation-state-specific recognition of histones by the MBT repeat protein L3MBTL2
Guo Y, Nady N, Qi C, et al. Methylation-state-specific recognition of histones by the MBT repeat protein L3MBTL2. Nucleic Acids Res 2009; 37: 2204-2210.
L3MBTL1 polycomb protein acandidate tumor suppressor in del(20q12) myeloid disorders is essential for genome stability
Gurvich N, Perna F, Farina A, et al. L3MBTL1 polycomb protein, acandidate tumor suppressor in del(20q12) myeloid disorders, is essential for genome stability. Proc Natl Acad Sci USA 2010; 107: 22552-22557.