Immunometabolism: IL-6, the resistance fighter

Nature Reviews Immunology

Volumn 14, Issue 5, 2014, Pages 282-293

  Bordon, Yvonne ^a  

^a NONE

Author keywords

[No Author keywords available]

Indexed keywords

INTERLEUKIN 4; INTERLEUKIN 4 RECEPTOR; INTERLEUKIN 6; INTERLEUKIN 6 RECEPTOR; INTERLEUKIN 6 RECEPTOR ALPHA; LEPTIN; LIPOPOLYSACCHARIDE; STRESS ACTIVATED PROTEIN KINASE; TRIACYLGLYCEROL;

BODY FAT; BODY WEIGHT; BONE MARROW CELL; BROWN ADIPOSE TISSUE; CIRCULATION; DIET; GENE; GENE EXPRESSION; GLUCONEOGENESIS; GLUCOSE INTOLERANCE; GLUCOSE TOLERANCE; GLUCOSE TRANSPORT; HORMONE BLOOD LEVEL; HUMAN; INFLAMMATION; INSULIN RESISTANCE; LIPID DIET; LIPOLYSIS; LIPOPOLYSACCHARIDE-INDUCED ENDOTOXEMIA; LIVER; MACROPHAGE; NONHUMAN; NOTE; OBESITY; PHENOTYPE; PRIORITY JOURNAL; TRIACYLGLYCEROL BLOOD LEVEL; WEIGHT GAIN; WHITE ADIPOSE TISSUE;

ANIMALS; ENDOTOXEMIA; HUMANS; INSULIN RESISTANCE; INTERLEUKIN-6; MACROPHAGE ACTIVATION; MACROPHAGES; OBESITY;

EID: 84899635771     PISSN: 14741733     EISSN: 14741741     Source Type: Journal    
DOI: 10.1038/nri3670     Document Type: Note

References (1)

1. Mauer, J. et al. Signaling by IL?6 promotes alternative activation of macrophages to limit endotoxemia and obesity-associated resistance to insulin. Nature Immunol. http://dx.doi.org/10-1038/ni.2865 (2014)