Autophagy, p53, and pancreatic cancer

New England Journal of Medicine

Volumn 370, Issue 14, 2014, Pages 1352-1353

  Iacobuzio Donahue, Christine A ^a   Herman, Joseph M ^b  

^a MEMORIAL SLOAN KETTERING CANCER CENTER   (United States)

^b JOHNS HOPKINS HOSPITAL   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

HYDROXYCHLOROQUINE; PROTEIN P53;

ARTICLE; AUTOPHAGOSOME; AUTOPHAGY; CARCINOGENESIS; DRUG EFFECT; GENE DELETION; GENE MUTATION; HUMAN; NONHUMAN; ONCOGENE K RAS; PANCREAS ADENOCARCINOMA; PANCREAS CANCER; PRIORITY JOURNAL; PROTEIN DEGRADATION; TUMOR SUPPRESSOR GENE;

ANIMALS; AUTOPHAGY; DISEASE MODELS, ANIMAL; HUMANS; MICE; MUTATION; PANCREATIC NEOPLASMS; PROTO-ONCOGENE PROTEINS; RAS PROTEINS; TUMOR SUPPRESSOR PROTEIN P53;

EID: 84897528091     PISSN: 00284793     EISSN: 15334406     Source Type: Journal    
DOI: 10.1056/NEJMcibr1400189     Document Type: Article

References (5)

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4. Klein AP, Lindström S, Mendelsohn JB, et al. An absolute risk model to identify individuals at elevated risk for pancreatic cancer in the general population. PLoS One 2013;8(9):e72311.
5. Yachida S, White CM, Naito Y, et al. Clinical significance of the genetic landscape of pancreatic cancer and implications for identification of potential long-term survivors. Clin Cancer Res 2012;18:6339-47.