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Volumn 73, Issue 3, 2014, Pages 261-264

Psoriasis-like skin lesions are dependent on IL-23 but develop in the absence of IL-22 in a model mouse

Author keywords

[No Author keywords available]

Indexed keywords

ANIMALS; DISEASE MODELS, ANIMAL; INTERLEUKIN-10; INTERLEUKIN-23; INTERLEUKINS; MICE; PSORIASIS;

EID: 84895090931     PISSN: 09231811     EISSN: 1873569X     Source Type: Journal    
DOI: 10.1016/j.jdermsci.2013.11.008     Document Type: Letter
Times cited : (9)

References (10)
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  • 2
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  • 3
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    • Distinct roles of IL-23 and IL-17 in the development of psoriasis-like lesions in a mouse model
    • Nakajima K., Kanda T., Takaishi M., Shiga T., Miyoshi K., Nakajima H., et al. Distinct roles of IL-23 and IL-17 in the development of psoriasis-like lesions in a mouse model. J Immunol 2011, 186:4481-4489.
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  • 4
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    • Interleukin (IL)-19, IL-20 and IL-24 are produced by and act on keratinocytes and are distinct from classical ILs
    • Kunz S., Wolk K., Witte E., Witte K., Doecke W.D., Volk H.D., et al. Interleukin (IL)-19, IL-20 and IL-24 are produced by and act on keratinocytes and are distinct from classical ILs. Exp Dermatol 2006, 15:991-1004.
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  • 5
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    • He M., Liang P. IL-24 transgenic mice: in vivo evidence of overlapping functions for IL-20, IL-22, and IL-24 in the epidermis. J Immunol 2010, 184:1793-1798.
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  • 6
    • 33751546237 scopus 로고    scopus 로고
    • IL-23 stimulates epidermal hyperplasia via TNF and IL-20R2-dependent mechanisms with implications for psoriasis pathogenesis
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.