Glyoxalase-1 overexpression in bone marrow cells reverses defective neovascularization in STZ-induced diabetic mice

Cardiovascular Research

Volumn 101, Issue 2, 2014, Pages 306-316

  Vulesevic, Branka ^a   McNeill, Brian ^a   Geoffrion, Michele ^a   Kuraitis, Drew ^a   McBane, Joanne E ^a   Lochhead, Marina ^a   Vanderhyden, Barbara C ^b   Korbutt, Gregory S ^c   Milne, Ross W ^a   Suuronen, Erik J ^a  

^a UNIVERSITY OF OTTAWA HEART INSTITUTE   (Canada)

^b OTTAWA HOSPITAL RESEARCH INSTITUTE   (Canada)

^c UNIVERSITY OF ALBERTA   (Canada)

Author keywords

Bone marrow cells; Methylglyoxal; Neovascularization; Type 1 diabetes

Indexed keywords

ENDOTHELIAL NITRIC OXIDE SYNTHASE; ENHANCED GREEN FLUORESCENT PROTEIN; GLYOXALASE; GLYOXALASE 1; MESSENGER RNA; PROTEIN BCL 2; PROTEIN BCL XL; STREPTOZOCIN; STROMAL CELL DERIVED FACTOR 1; TUMOR NECROSIS FACTOR ALPHA; UNCLASSIFIED DRUG; VASCULOTROPIN; VASCULOTROPIN A;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTERIOLE; ARTICLE; BONE MARROW CELL; CAPILLARY DENSITY; CELL HOMING; CELL MIGRATION; CELL VIABILITY; COMPARATIVE STUDY; CONTROLLED STUDY; ENDOTHELIUM CELL; ENZYME ACTIVITY; GENE OVEREXPRESSION; HYPERGLYCEMIA; HYPOXIA; LIMB ISCHEMIA; LIMB PERFUSION; MACROPHAGE; MALE; MOUSE; NEOVASCULARIZATION (PATHOLOGY); NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; SMOOTH MUSCLE FIBER; STREPTOZOTOCIN-INDUCED DIABETES MELLITUS; SURVIVAL; TRANSGENE; WILD TYPE;

BONE MARROW CELLS; METHYLGLYOXAL; NEOVASCULARIZATION; TYPE 1 DIABETES;

ANGIOGENIC PROTEINS; ANIMALS; APOPTOSIS; BLOOD GLUCOSE; BONE MARROW CELLS; BONE MARROW TRANSPLANTATION; CELL MOVEMENT; CELL SURVIVAL; CELLS, CULTURED; CYTOKINES; DIABETES MELLITUS, EXPERIMENTAL; DIABETES MELLITUS, TYPE 1; DIABETIC ANGIOPATHIES; HINDLIMB; HUMANS; ISCHEMIA; LACTOYLGLUTATHIONE LYASE; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; MUSCLE, SKELETAL; NEOVASCULARIZATION, PATHOLOGIC; NITRIC OXIDE SYNTHASE TYPE III; OXIDATION-REDUCTION; OXIDATIVE STRESS; PYRUVALDEHYDE; RECOVERY OF FUNCTION; REGIONAL BLOOD FLOW; TIME FACTORS; UP-REGULATION;

EID: 84893090584     PISSN: 00086363     EISSN: 17553245     Source Type: Journal    
DOI: 10.1093/cvr/cvt259     Document Type: Article

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