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Volumn , Issue , 2009, Pages 71-74

Fetal and Neonatal Bone Development

Author keywords

Bone development; Calcium; Newborn; Parathyroid hormone; Vitamin D

Indexed keywords


EID: 84889363781     PISSN: None     EISSN: None     Source Type: Book    
DOI: 10.1002/9780470623992.ch13     Document Type: Chapter
Times cited : (6)

References (20)
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  • 2
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  • 3
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  • 4
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    • Human fetal bone development: histomorphometric evaluation of the proximal femoral metaphysis
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    • (2002) Bone , vol.30 , pp. 823-828
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  • 5
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    • Sequential changes in weight, density, and percentage ash weight of human skeletons from an early fetal period through old age
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    • Trotter, M.1    Hixon, B.B.2
  • 6
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    • Maternal-fetal calcium and bone metabolism during pregnancy, puerperium, and lactation
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  • 7
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    • Absence of mechanical loading in utero influences bone mass and architecture but not innervation in Mvod-Myf5-deficient mice
    • Gomez C, David V, Peet NM, Vico L, Chenu C. Malaval L, Skerry TM 2007 Absence of mechanical loading in utero influences bone mass and architecture but not innervation in Mvod-Myf5-deficient mice. J Anat 210:259-271.
    • (2007) J Anat , vol.210 , pp. 259-271
    • Gomez, C.1    David, V.2    Peet, N.M.3    Vico, L.4    Chenu, C.5    Malaval, L.6    Skerry, T.M.7
  • 9
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    • The developing bone: Slave or master of its cells and molecules?
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  • 11
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    • Biochemical markers of bone metabolism
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    • Schoenau E, Rauch F 2003 Biochemical markers of bone metabolism. In: Glorieux FH, Pettifor J, Jueppner H (eds.) Pediatric Bone. Academic Press, San Diego, CA, USA, pp. 339-357.
    • (2003) Pediatric Bone , pp. 339-357
    • Schoenau, E.1    Rauch, F.2
  • 12
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    • Changes in bone density during childhood and adolescence: An approach based on bone's biological organization
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    • Rauch, F.1    Schoenau, E.2
  • 13
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    • In vivo and in vitro characterization of neonatal hyperparathyroidism resulting from a de novo, heterozygous mutation in the Ca2+-sensing receptor gene: Normal maternal calcium homeostasis as a cause of secondary hyperparathyroidism in familial benign hypocalciuric hypercalcemia
    • Bai M. Pearce SH, Kifor O, Trivedi S, Stauffer UG, Thakker RV, Brown EM, Steinmann B 1997 In vivo and in vitro characterization of neonatal hyperparathyroidism resulting from a de novo, heterozygous mutation in the Ca2+-sensing receptor gene: Normal maternal calcium homeostasis as a cause of secondary hyperparathyroidism in familial benign hypocalciuric hypercalcemia. J Clin Invest 99:88-96.
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    • Bai, M.1    Pearce, S.H.2    Kifor, O.3    Trivedi, S.4    Stauffer, U.G.5    Thakker, R.V.6    Brown, E.M.7    Steinmann, B.8
  • 16
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    • Vitamin D requirements during pregnancy
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  • 19
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    • Unexplained fractures in infancy: Looking for fragile bones
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.