Copper-induced alterations in rat brain depends on route of overload and basal copper levels

Nutrition

Volumn 30, Issue 1, 2014, Pages 96-106

  Arnal, Nathalie ^a   Dominici, Lina ^a   Tacconi, María J T de ^a   Marra, Carlos Alberta ^a  

^a INSTITUTO DE INVESTIGACIONES BIOQUÍMICAS DE LA PLATA INIBIOLP   (Argentina)

Author keywords

Apoptosis; Brain; Copper; Lipids; Oxidative stress

Indexed keywords

CALPAIN; CASPASE 3; CERULOPLASMIN; COPPER; METALLOTHIONEIN;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; BRAIN LEVEL; BRAIN METABOLISM; CELL SURVIVAL; CONCENTRATION (PARAMETERS); CONTROLLED STUDY; COPPER BLOOD LEVEL; COPPER METABOLISM; ENZYME ACTIVITY; MALE; NEUROTOXICITY; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; RAT;

APOPTOSIS; BRAIN; COPPER; LIPIDS; OXIDATIVE STRESS;

ADMINISTRATION, ORAL; ANIMALS; BIOLOGICAL MARKERS; BRAIN; CARBONATES; CASPASE 3; COPPER; DOSE-RESPONSE RELATIONSHIP, DRUG; INJECTIONS, INTRAPERITONEAL; LIPID METABOLISM; MALE; OXIDATIVE STRESS; RATS; RATS, WISTAR; REACTIVE OXYGEN SPECIES;

EID: 84888403858     PISSN: 08999007     EISSN: 18731244     Source Type: Journal    
DOI: 10.1016/j.nut.2013.06.009     Document Type: Article

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