T-bet is essential for Th1-mediated, but not Th17-mediated, CNS autoimmune disease

European Journal of Immunology

Volumn 43, Issue 11, 2013, Pages 2818-2823

  O'Connor, Richard A ^a   Cambrook, Helen ^a   Huettner, Katja ^a   Anderton, Stephen M ^a  

^a UNIVERSITY OF EDINBURGH   (United Kingdom)

Author keywords

EAE; T cells; T bet; Th1; Th17

Indexed keywords

GAMMA INTERFERON; GRANULOCYTE MACROPHAGE COLONY STIMULATING FACTOR; INTERLEUKIN 17; INTERLEUKIN 23; INTERLEUKIN 6; MYELIN OLIGODENDROCYTE GLYCOPROTEIN; TRANSCRIPTION FACTOR T BET; TRANSFORMING GROWTH FACTOR BETA;

ADOPTIVE TRANSFER; ALLERGIC ENCEPHALOMYELITIS; ANIMAL EXPERIMENT; ARTICLE; CD4+ T LYMPHOCYTE; CYTOKINE PRODUCTION; IMMUNOREACTIVITY; IN VITRO STUDY; INFLAMMATION; MOUSE; NONHUMAN; PATHOGENICITY; PRIORITY JOURNAL; TH1 CELL; TH17 CELL;

EAE; T CELLS; T-BET; TH1; TH17;

ANIMALS; AUTOIMMUNITY; CELL DIFFERENTIATION; CELLS, CULTURED; CENTRAL NERVOUS SYSTEM; ENCEPHALOMYELITIS, AUTOIMMUNE, EXPERIMENTAL; GRANULOCYTE-MACROPHAGE COLONY-STIMULATING FACTOR; INFLAMMATION; INTERFERON-GAMMA; INTERLEUKIN-17; INTERLEUKIN-23; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; NUCLEAR RECEPTOR SUBFAMILY 1, GROUP F, MEMBER 3; T-BOX DOMAIN PROTEINS; TH1 CELLS; TH17 CELLS;

EID: 84888016708     PISSN: 00142980     EISSN: 15214141     Source Type: Journal    
DOI: 10.1002/eji.201343689     Document Type: Article

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