Ischemic post-conditioning facilitates brain recovery after stroke by promoting Akt/mTOR activity in nude rats

Journal of Neurochemistry

Volumn 127, Issue 5, 2013, Pages 723-732

  Xie, Rong ^a,b,c   Wang, Peng ^a,d   Ji, Xunming ^d   Zhao, Heng ^a  

^a STANFORD UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b FUDAN UNIVERSITY   (China)

^c Shanghai Jingan District Central Hospital   (China)

^d CAPITAL MEDICAL UNIVERSITY   (China)

Author keywords

Akt; ischemic post conditioning; mTOR; stroke; T cells

Indexed keywords

AKT1 PROTEIN; AKT3 PROTEIN; INITIATION FACTOR 4E BINDING PROTEIN 1; ISOENZYME; MAMMALIAN TARGET OF RAPAMYCIN; NEUROMODULIN; PROTEIN KINASE B; PROTEIN KINASE B BETA; S6 KINASE; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN INFARCTION SIZE; CEREBROVASCULAR ACCIDENT; CONTROLLED STUDY; CONVALESCENCE; ENZYME ACTIVITY; ENZYME INHIBITION; ENZYME MECHANISM; ENZYME PHOSPHORYLATION; ISCHEMIC POSTCONDITIONING; MALE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT; SIGNAL TRANSDUCTION;

ANIMALIA; MAMMALIA; RATTUS;

AKT; ISCHEMIC POST-CONDITIONING; MTOR; STROKE; T CELLS;

ANIMALS; DISEASE MODELS, ANIMAL; IMMUNOSUPPRESSIVE AGENTS; ISCHEMIC POSTCONDITIONING; MALE; PROTO-ONCOGENE PROTEINS C-AKT; RATS; RATS, NUDE; RATS, SPRAGUE-DAWLEY; RECOVERY OF FUNCTION; RIBOSOMAL PROTEIN S6 KINASES, 70-KDA; SIROLIMUS; STROKE; T-LYMPHOCYTES; TOR SERINE-THREONINE KINASES;

EID: 84887618035     PISSN: 00223042     EISSN: 14714159     Source Type: Journal    
DOI: 10.1111/jnc.12342     Document Type: Article

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