Helicobacter pylori induces type 4 secretion system-dependent, but CagA-independent activation of IκBs and NF-κB/RelA at early time points

International Journal of Medical Microbiology

Volumn 303, Issue 8, 2013, Pages 548-552

  Sokolova, Olga ^a   Borgmann, Marc ^a   Rieke, Cornelia ^a   Schweitzer, Katrin ^a   Rothkötter, Hermann Josef ^a   Naumann, Michael ^a  

^a OTTO VON GUERICKE UNIVERSITY   (Germany)

Author keywords

CagPAI pathogenicity island; I Bs; Inflammation; RelA phosphorylation

Indexed keywords

CAGA PROTEIN; I KAPPA B; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; INTERLEUKIN 8; TRANSCRIPTION FACTOR RELA;

ARTICLE; BACTERIAL STRAIN; CONTROLLED STUDY; DEGRADATION; DNA BINDING; GENE ACTIVATION; GENE EXPRESSION; HELICOBACTER PYLORI; NONHUMAN; PROTEIN PHOSPHORYLATION; TYPE IV SECRETION SYSTEM;

CAGPAI PATHOGENICITY ISLAND; INFLAMMATION; IΚBS; RELA PHOSPHORYLATION;

ANTIGENS, BACTERIAL; BACTERIAL PROTEINS; BACTERIAL SECRETION SYSTEMS; GENE SILENCING; HELICOBACTER PYLORI; HUMANS; NF-KAPPA B; PHOSPHORYLATION; PROTEIN PROCESSING, POST-TRANSLATIONAL; TRANSCRIPTIONAL ACTIVATION;

EID: 84887613180     PISSN: 14384221     EISSN: 16180607     Source Type: Journal    
DOI: 10.1016/j.ijmm.2013.07.008     Document Type: Article

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