Reply to Osterburg et al.: To study human inflammatory diseases in humans

Proceedings of the National Academy of Sciences of the United States of America

Volumn 110, Issue 36, 2013, Pages

  Tompkins, Ronald G ^a   Warren, H Shaw ^a   Mindrinos, Michael N ^b   Xiao, Wenzhong ^a,b   Davis, Ronald W ^a  

^a MASSACHUSETTS GENERAL HOSPITAL   (United States)

^b STANFORD UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

LIPOPOLYSACCHARIDE;

CELL POPULATION; CROSS-SECTIONAL STUDY; HUMAN; INFLAMMATORY DISEASE; LETTER; LEUKOCYTE; NONHUMAN; PRIORITY JOURNAL; RESPONSE TIME; ANIMAL; FEMALE; GENETICS; GENOMICS; INFLAMMATION; MALE;

ANIMALS; FEMALE; GENOMICS; HUMANS; INFLAMMATION; MALE;

EID: 84883345596     PISSN: 00278424     EISSN: 10916490     Source Type: Journal    
DOI: 10.1073/pnas.1307452110     Document Type: Letter

References (4)

1. Osterburg AR, et al. (2013) Concerns over interspecies transcriptional comparisons in mice and humans after trauma. Proc Natl Acad Sci USA 110:E3370.
2. Seok J, et al.; Inflammation and Host Response to Injury, Large Scale Collaborative Research Program (2013) Genomic responses in mouse models poorly mimic human inflammatory diseases. Proc Natl Acad Sci USA 110(9):3507-3512.
3. De Maio A, Mooney ML, Matesic LE, Paidas CN, Reeves RH (1998) Genetic component in the inflammatory response induced by bacterial lipopolysaccharide. Shock 10(5):319-323. (Pubitemid 128463268)
4. Storey JD, Xiao W, Leek JT, Tompkins RG, Davis RW (2005) Significance analysis of time course microarray experiments. Proc Natl Acad Sci USA 102(36):12837-12842. (Pubitemid 41279452)