Comment on: Teeuwisse et al. Short-term caloric restriction normalizes hypothalamic neuronal responsiveness to glucose ingestion in patients with type 2 diabetes. Diabetes 2012;61:3255-3259

Diabetes

Volumn 62, Issue 6, 2013, Pages

  Heni, Martin ^a   Kullmann, Stephanie ^a   Fritsche, Andreas ^a  

^a UNIVERSITY OF TÜBINGEN   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

GLUCOSE; INSULIN;

BRAIN METABOLISM; CALORIC RESTRICTION; FUNCTIONAL MAGNETIC RESONANCE IMAGING; HUMAN; HYPOTHALAMUS NUCLEUS; INSULIN SENSITIVITY; LOW CALORY DIET; NERVE CELL; NON INSULIN DEPENDENT DIABETES MELLITUS; NOTE; ORAL GLUCOSE TOLERANCE TEST; PRIORITY JOURNAL;

CALORIC RESTRICTION; DIABETES MELLITUS, TYPE 2; GLUCOSE; HUMANS; HYPOTHALAMUS; MALE;

EID: 84878236411     PISSN: 00121797     EISSN: 1939327X     Source Type: Journal    
DOI: 10.2337/db13-0054     Document Type: Note

References (9)

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2. Vidarsdottir S, Smeets PAM, Eichelsheim DL, et al. Glucose ingestion fails to inhibit hypothalamic neuronal activity in patients with type 2 diabetes. Diabetes 2007;56:2547-2550 (Pubitemid 47523262)
3. Smeets PAM, de Graaf C, Sta fleu A, van Osch MJP, van der Grond J. Functional MRI of human hypothalamic responses following glucose ingestion. Neuroimage 2005;24:363-368 (Pubitemid 41359033)
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9. Tschritter O, Preissl H, Hennige AM, et al. High cerebral insulin sensitivity is associated with loss of body fat during lifestyle intervention. Diabetologia 2012;55:175-182