All tyrosine kinase inhibitor-resistant chronic myelogenous cells are highly sensitive to Ponatinib

Oncotarget

Volumn 3, Issue 12, 2012, Pages 1557-1565

  Cassuto, Ophélie ^a,d   Dufies, Maeva ^a,b   Jacquel, Arnaud ^a,b   Robert, Guillaume ^a,b   Ginet, Clémence ^a,b   Dubois, Alix ^a,b   Hamouda, Amine ^a,b   Puissant, Alexandre ^a,b,e   Luciano, Fredéric ^a,b   Karsenti, Jean Michel ^c   Legros, Laurence ^c   Cassuto, Jill Patrice ^c   Lenain, Pascal ^d   Auberger, Patrick ^a,c  

^a CENTRE MÉDITERRANÉEN DE MÉDECINE MOLÉCULAIRE C3M   (France)

^b Equipe Labellisée 2007 Ligue Nationale Contre le Cancer   (France)

^c UNIVERSITY HOSPITAL OF NICE   (France)

^d CENTRE HENRI BECQUEREL   (France)

^e DANA FARBER CANCER INSTITUTE   (United States)

Author keywords

BCR ABL; CML; Ponatinib; Resistance; TKI

Indexed keywords

BCR ABL PROTEIN; DASATINIB; IMATINIB; PONATINIB; PROTEIN TYROSINE KINASE INHIBITOR; THYMINE;

ANIMAL CELL; APOPTOSIS; ARTICLE; B LYMPHOCYTE; CANCER PATIENT; CANCER RESISTANCE; CELL STRAIN; CELL STRAIN BAF3; CELL VIABILITY; CHEMOSENSITIVITY; CHRONIC MYELOID LEUKEMIA; CONTROLLED STUDY; DRUG EFFECT; HUMAN; HUMAN CELL; LEUKEMIA CELL; LEUKEMIA CELL LINE; MOUSE; MUTATION; NONHUMAN; SIGNAL TRANSDUCTION;

EID: 84874715679     PISSN: None     EISSN: 19492553     Source Type: Journal    
DOI: 10.18632/oncotarget.692     Document Type: Article

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