In vivo suppression of microRNA-24 prevents the transition toward decompensated hypertrophy in aortic-constricted mice

Circulation Research

Volumn 112, Issue 4, 2013, Pages 601-605

  Li, Rong Chang ^a   Tao, Jin ^a   Guo, Yun Bo ^a   Wu, Hao Di ^a   Liu, Rui Feng ^a   Bai, Yan ^a   Lv, Zhi Zhen ^a   Luo, Guan Zheng ^b   Li, Lin Lin ^a   Wang, Meng ^b   Yang, Hua Qian ^a   Gao, Wei ^a   Han, Qi De ^a   Zhang, You Yi ^a   Wang, Xiu Jie ^b   Xu, Ming ^a   Wang, Shi Qiang ^a  

^a PEKING UNIVERSITY   (China)

^b INSTITUTE OF GENETICS AND DEVELOPMENTAL BIOLOGY   (China)

Author keywords

Ca2+ signaling; heart failure; hypertrophic cardiomyopathy; hypertrophy; myocardial contraction

Indexed keywords

CALCIUM CHANNEL; JUNCTOPHILIN 2 PROTEIN; MICRORNA 24; PROTEIN; RYANODINE RECEPTOR; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL MODEL; ARTICLE; ELECTROPHYSIOLOGY; HEART CONTRACTION; HEART HYPERTROPHY; HEART MUSCLE CELL; IN VIVO STUDY; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; SARCOPLASMIC RETICULUM;

ANIMALS; AORTIC STENOSIS, SUBVALVULAR; CALCIUM CHANNELS, L-TYPE; CALCIUM SIGNALING; DISEASE PROGRESSION; DRUG EVALUATION, PRECLINICAL; EXCITATION CONTRACTION COUPLING; GENE EXPRESSION REGULATION; HEART FAILURE; HYPERTROPHY, LEFT VENTRICULAR; MALE; MEMBRANE PROTEINS; MICE; MICE, INBRED C57BL; MICRORNAS; MODELS, CARDIOVASCULAR; MYOCARDIAL CONTRACTION; MYOCYTES, CARDIAC; OLIGONUCLEOTIDES, ANTISENSE; RYANODINE RECEPTOR CALCIUM RELEASE CHANNEL; SARCOPLASMIC RETICULUM;

EID: 84874255537     PISSN: 00097330     EISSN: 15244571     Source Type: Journal    
DOI: 10.1161/CIRCRESAHA.112.300806     Document Type: Article

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