VDAC: Old protein with new roles in diabetes

American Journal of Physiology - Cell Physiology

Volumn 303, Issue 10, 2012, Pages

  Sasaki, Koh ^a   Donthamsetty, Reshma ^a   Heldak, Michael ^a   Cho, Young Eun ^a   Scott, Brian T ^b   Makino, Ayako ^a  

^a UNIVERSITY OF ILLINOIS AT CHICAGO   (United States)

^b UNIVERSITY OF CALIFORNIA SAN DIEGO   (United States)

Author keywords

Apoptosis; Ca2+ overload in mitochondria; Vascular complications; Vascular rarefaction

Indexed keywords

CALCIUM ION; MITOCHONDRIAL PERMEABILITY TRANSITION PORE; OXYGEN; SHORT HAIRPIN RNA; SUPEROXIDE; VOLTAGE DEPENDENT ANION CHANNEL;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; APOPTOSIS; ARTICLE; CALCIUM TRANSPORT; CAPILLARY DENSITY; CONTROLLED STUDY; DOWN REGULATION; ENDOTHELIAL DYSFUNCTION; ENDOTHELIUM CELL; HEART LEFT VENTRICLE; HEART MUSCLE ISCHEMIA; MALE; MITOCHONDRION; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN BLOOD LEVEL; PROTEIN FUNCTION; STREPTOZOCIN DIABETES;

ANIMALS; APOPTOSIS; CALCIUM; CORONARY VESSELS; DIABETES MELLITUS, EXPERIMENTAL; ENDOTHELIAL CELLS; GENE EXPRESSION REGULATION; HEART VENTRICLES; HEXOKINASE; MALE; MICE; MICE, INBRED C57BL; MITOCHONDRIA; MYOCARDIUM; REACTIVE OXYGEN SPECIES; RNA, SMALL INTERFERING; VOLTAGE-DEPENDENT ANION CHANNELS;

MUS;

EID: 84869219148     PISSN: 03636143     EISSN: 15221563     Source Type: Journal    
DOI: 10.1152/ajpcell.00087.2012     Document Type: Article

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