Zebrafish model for congenital myasthenic syndrome reveals mechanisms causal to developmental recovery

Proceedings of the National Academy of Sciences of the United States of America

Volumn 109, Issue 43, 2012, Pages 17711-17716

  Walogorsky, Michael ^a   Mongeon, Rebecca ^a   Wen, Hua ^a   Nelson, Nathan R ^a   Urban, Jason M ^b   Ono, Fumihito ^b   Mandel, Gail ^a   Brehm, Paul ^a  

^a OREGON HEALTH AND SCIENCE UNIVERSITY   (United States)

^b NATIONAL INSTITUTE ON ALCOHOL ABUSE AND ALCOHOLISM   (United States)

Author keywords

Neuromuscular; Nicotinic receptor; Quinidine

Indexed keywords

ISOPROTEIN;

ADULTHOOD; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CONGENITAL MYASTHENIC SYNDROME; CONTROLLED STUDY; DEVELOPMENTAL STAGE; DISEASE ASSOCIATION; DISEASE MODEL; EMBRYO; EMBRYO DEVELOPMENT; FORCED SWIMMING TEST; LARVA; MEMBRANE DEPOLARIZATION; MUTATIONAL ANALYSIS; NERVOUS SYSTEM ELECTROPHYSIOLOGY; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; QUANTITATIVE ANALYSIS; VOLTAGE CLAMP; WILD TYPE; ZEBRA FISH;

ANIMALS; BASE SEQUENCE; DISEASE MODELS, ANIMAL; DNA PRIMERS; MYASTHENIC SYNDROMES, CONGENITAL; PATCH-CLAMP TECHNIQUES; ZEBRAFISH;

EID: 84867907512     PISSN: 00278424     EISSN: 10916490     Source Type: Journal    
DOI: 10.1073/pnas.1215858109     Document Type: Article

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