Tumorigenesis: Pushing pancreatic cancer to take off

Nature Reviews Cancer

Volumn 12, Issue 11, 2012, Pages 739-

  Seton Rogers, Sarah ^a  

^a NONE

Author keywords

[No Author keywords available]

Indexed keywords

ADAM PROTEIN; AMPHIREGULIN; EPIDERMAL GROWTH FACTOR RECEPTOR; ERLOTINIB; GEMCITABINE; STAT3 PROTEIN; TRANSFORMING GROWTH FACTOR ALPHA;

ACINAR CELL; CANCER RISK; CANCER SURVIVAL; CARCINOGENESIS; CHRONIC PANCREATITIS; DISEASE COURSE; EXPLANT; GENE; GENE EXPRESSION; HUMAN; KRAS GENE; METAPLASIA; NONHUMAN; NOTE; PANCREAS ADENOCARCINOMA; PANCREAS ADENOMA; PRIORITY JOURNAL; SIGNAL TRANSDUCTION; TUMOR SUPPRESSOR GENE; UPREGULATION;

EID: 84867881542     PISSN: 1474175X     EISSN: 14741768     Source Type: Journal    
DOI: 10.1038/nrc3383     Document Type: Note

References (2)

1. Ardito, C. M. et al. EGF receptor is required for KRAS-induced pancreatic tumorigenesis. Cancer Cell 22, 304-317 (2012)
2. Navas, C. et al. EGF receptor signaling is essential for K-Ras oncogene-driven pancreatic ductal adenocarcinoma. Cancer Cell 22, 318-330 (2012)