A soluble receptor for advanced glycation end-products inhibits hypoxia/reoxygenation-induced apoptosis in rat cardiomyocytes via the mitochondrial pathway

International Journal of Molecular Sciences

Volumn 13, Issue 9, 2012, Pages 11923-11940

  Guo, Caixia ^a   Zeng, Xiangjun ^b   Song, Juanjuan ^a   Zhang, Min ^a   Wang, Hongxia ^b   Xu, Xiaowei ^a   Du, Fenghe ^a   Chen, Buxing ^a  

^a CAPITAL MEDICAL UNIVERSITY   (China)

^b CAPITAL MEDICAL UNIVERSITY   (China)

Author keywords

Apoptosis; Ischemia; Mitochondria; Reperfusion; sRAGE

Indexed keywords

ADVANCED GLYCATION END PRODUCT RECEPTOR; CASPASE 3; CYTOCHROME C; MITOCHONDRIAL PERMEABILITY TRANSITION PORE; PROTEIN BAX; PROTEIN BCL 2;

ANIMAL CELL; ANTIPROLIFERATIVE ACTIVITY; APOPTOSIS; ARTICLE; CELL MEMBRANE DEPOLARIZATION; CELL VIABILITY; CONTROLLED STUDY; DRUG EFFECT; DRUG MECHANISM; ENZYME ACTIVITY; HEART MUSCLE CELL; HEART MUSCLE ISCHEMIA; HEART PROTECTION; IN VITRO STUDY; MITOCHONDRIAL MEMBRANE POTENTIAL; NEWBORN; NONHUMAN; PROTEIN SECRETION; RAT; REPERFUSION INJURY; TREATMENT OUTCOME;

RATTUS;

EID: 84866905191     PISSN: 16616596     EISSN: 14220067     Source Type: Journal    
DOI: 10.3390/ijms130911923     Document Type: Article

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