-
1
-
-
1542547460
-
Identification of telmisartan as a unique angiotensin II receptor antagonist with selective PPARgammamodulating activity
-
Benson SC, Pershadsingh HA, Ho CI, et al. Identification of telmisartan as a unique angiotensin II receptor antagonist with selective PPARgammamodulating activity. Hypertension. 2004;43:993-1002.
-
(2004)
Hypertension
, vol.43
, pp. 993-1002
-
-
Benson, S.C.1
Pershadsingh, H.A.2
Ho, C.I.3
-
2
-
-
11144313062
-
Telmisartan is a promising cardiometabolic sartan due to its unique PPAR-gamma-inducing property
-
Yamagishi S, Takeuchi M. Telmisartan is a promising cardiometabolic sartan due to its unique PPAR-gamma-inducing property. Med hypotheses. 2005;64:476-478.
-
(2005)
Med Hypotheses
, vol.64
, pp. 476-478
-
-
Yamagishi, S.1
Takeuchi, M.2
-
3
-
-
33646148948
-
Telmisartan but not valsartan increases caloric expenditure and protects against weight gain and hepatic steatosis
-
Sugimoto K, Qi NR, Kazdova L, et al. Telmisartan but not valsartan increases caloric expenditure and protects against weight gain and hepatic steatosis. Hypertension. 2006;47:1003-1009.
-
(2006)
Hypertension
, vol.47
, pp. 1003-1009
-
-
Sugimoto, K.1
Qi, N.R.2
Kazdova, L.3
-
4
-
-
2442451618
-
Angiotensin type 1 receptor blockers induce peroxisome proliferator-activated receptor-gamma activity
-
Schupp M, Janke J, Clasen R, et al. Angiotensin type 1 receptor blockers induce peroxisome proliferator-activated receptor-gamma activity. Circulation. 2004;109:2054-2057.
-
(2004)
Circulation
, vol.109
, pp. 2054-2057
-
-
Schupp, M.1
Janke, J.2
Clasen, R.3
-
5
-
-
42549153011
-
Cardioprotective mechanism of telmisartan via PPAR-gamma-eNOS pathway in dahl salt-sensitive hypertensive rats
-
Kobayashi N, Ohno T, Yoshida K, et al. Cardioprotective mechanism of telmisartan via PPAR-gamma-eNOS pathway in dahl salt-sensitive hypertensive rats. Am J Hypertens. 2008;21:576-581.
-
(2008)
Am J Hypertens
, vol.21
, pp. 576-581
-
-
Kobayashi, N.1
Ohno, T.2
Yoshida, K.3
-
6
-
-
51449089179
-
Telmisartan inhibits advanced glycation end products (AGEs)-elicited endothelial cell injury by suppressing AGE receptor (RAGE) expression via peroxisome proliferator-activated receptor-gammaactivation
-
Yamagishi S, Matsui T, Nakamura K, et al. Telmisartan inhibits advanced glycation end products (AGEs)-elicited endothelial cell injury by suppressing AGE receptor (RAGE) expression via peroxisome proliferator-activated receptor-gammaactivation. Protein Pept Lett. 2008; 15:850-853.
-
(2008)
Protein Pept Lett
, vol.15
, pp. 850-853
-
-
Yamagishi, S.1
Matsui, T.2
Nakamura, K.3
-
7
-
-
43449087025
-
Telmisartan increases fatty acid oxidation in skeletal muscle through a peroxisome proliferator-activated receptor-gamma dependent pathway
-
Sugimoto K, Kazdova L, Qi NR, et al. Telmisartan increases fatty acid oxidation in skeletal muscle through a peroxisome proliferator-activated receptor-gamma dependent pathway. J Hypertens. 2008;26:1209-1215.
-
(2008)
J Hypertens
, vol.26
, pp. 1209-1215
-
-
Sugimoto, K.1
Kazdova, L.2
Qi, N.R.3
-
8
-
-
41649105085
-
Agonists at PPAR-gamma suppress angiotensin ii-induced production of plasminogen activator inhibitor-1 and extracellular matrix in rat cardiac fibroblasts
-
Hao GH, Niu XL, Gao DF, et al. Agonists at PPAR-gamma suppress angiotensin ii-induced production of plasminogen activator inhibitor-1 and extracellular matrix in rat cardiac fibroblasts. Br J Pharmacol. 2008;153:1409-1419.
-
(2008)
Br J Pharmacol
, vol.153
, pp. 1409-1419
-
-
Hao, G.H.1
Niu, X.L.2
Gao, D.F.3
-
9
-
-
77952295945
-
Modulation of PPAR-gamma by telmisartan protects the heart against myocardial infarction in experimental diabetes
-
Goyal S, Arora S, Bhatt TK, et al. Modulation of PPAR-gamma by telmisartan protects the heart against myocardial infarction in experimental diabetes. Chem Biol Interact. 2010;185:271-280.
-
(2010)
Chem Biol Interact
, vol.185
, pp. 271-280
-
-
Goyal, S.1
Arora, S.2
Bhatt, T.K.3
-
10
-
-
77955171963
-
Nonhypotensive dose of telmisartan attenuates cognitive impairment partially due to peroxisome proliferator-activated receptor-gamma activation in mice with chronic cerebral hypoperfusion
-
Washida K, Ihara M, Nishio K, et al. Nonhypotensive dose of telmisartan attenuates cognitive impairment partially due to peroxisome proliferator- activated receptor-gamma activation in mice with chronic cerebral hypoperfusion. Stroke. 2010;41:1798-1806.
-
(2010)
Stroke
, vol.41
, pp. 1798-1806
-
-
Washida, K.1
Ihara, M.2
Nishio, K.3
-
11
-
-
0032029246
-
Long acting calcium antagonist amlodipine prevents left ventricular remodeling after myocardial infarction in rats
-
Shimada T, Yoshiyama M, Takeuchi K, et al. Long acting calcium antagonist amlodipine prevents left ventricular remodeling after myocardial infarction in rats. Cardiovasc Res. 1998;37:618-626.
-
(1998)
Cardiovasc Res
, vol.37
, pp. 618-626
-
-
Shimada, T.1
Yoshiyama, M.2
Takeuchi, K.3
-
12
-
-
55949135607
-
Prevention of salt-induced hypertension and fibrosis by AT1-receptor blockers in Dahl S rats
-
Liang B, Leenen FH. Prevention of salt-induced hypertension and fibrosis by AT1-receptor blockers in Dahl S rats. J Cardiovasc Pharmacol. 2008;51:457-466.
-
(2008)
J Cardiovasc Pharmacol
, vol.51
, pp. 457-466
-
-
Liang, B.1
Leenen, F.H.2
-
13
-
-
0025978369
-
Cellular basis of chronic ventricular remodeling after myocardial infarction in rats
-
Olivetti G, Capasso JM, Meggs LG, et al. Cellular basis of chronic ventricular remodeling after myocardial infarction in rats. Circ Res. 1991;68:856-869.
-
(1991)
Circ Res
, vol.68
, pp. 856-869
-
-
Olivetti, G.1
Capasso, J.M.2
Meggs, L.G.3
-
14
-
-
0026316218
-
Cellular basis of ventricular remodeling after myocardial infarction
-
Anversa P, Olivetti G, Capasso JM. Cellular basis of ventricular remodeling after myocardial infarction. Am J Cardiol. 1991;68:7D-16D.
-
(1991)
Am J Cardiol
, vol.68
-
-
Anversa, P.1
Olivetti, G.2
Capasso, J.M.3
-
15
-
-
0031733939
-
Molecular structure and function of angiotensin ii receptors
-
Clauser E. Molecular structure and function of angiotensin ii receptors. Nephrologie. 1998;19:403-410.
-
(1998)
Nephrologie.
, vol.19
, pp. 403-410
-
-
Clauser, E.1
-
16
-
-
0031969613
-
Divergent effects of angiotensinconverting enzyme inhibition and angiotensin ii-receptor antagonism on myocardial cellular proliferation and collagen deposition after myocardial infarction in rats
-
Taylor K, Patten RD, Smith JJ, et al. Divergent effects of angiotensinconverting enzyme inhibition and angiotensin ii-receptor antagonism on myocardial cellular proliferation and collagen deposition after myocardial infarction in rats. J Cardiovasc Pharmacol. 1998;31:654-660.
-
(1998)
J Cardiovasc Pharmacol
, vol.31
, pp. 654-660
-
-
Taylor, K.1
Patten, R.D.2
Smith, J.J.3
-
17
-
-
14644435713
-
Targeted deletion or pharmacological inhibition of MMP-2 prevents cardiac rupture after myocardial infarction in mice
-
Matsumura S, Iwanaga S, Mochizuki S, et al. Targeted deletion or pharmacological inhibition of MMP-2 prevents cardiac rupture after myocardial infarction in mice. J Clin Invest. 2005;115:599-609.
-
(2005)
J Clin Invest
, vol.115
, pp. 599-609
-
-
Matsumura, S.1
Iwanaga, S.2
Mochizuki, S.3
-
18
-
-
34147160557
-
Cardiac matrix metalloproteinase- 2 expression independently induces marked ventricular remodeling and systolic dysfunction
-
Bergman MR, Teerlink JR, Mahimkar R, et al. Cardiac matrix metalloproteinase- 2 expression independently induces marked ventricular remodeling and systolic dysfunction. Am J Physiol Heart Circ Physiol. 2007;292:H1847-H1860.
-
(2007)
Am J Physiol Heart Circ Physiol.
, vol.292
-
-
Bergman, M.R.1
Teerlink, J.R.2
Mahimkar, R.3
-
19
-
-
31344477231
-
Relevance of matrix metalloproteinases and their inhibitors after myocardial infarction: A temporal and spatial window
-
Vanhoutte D, Schellings M, Pinto Y, et al. Relevance of matrix metalloproteinases and their inhibitors after myocardial infarction: a temporal and spatial window. Cardiovasc Res. 2006;69:604-613.
-
(2006)
Cardiovasc Res
, vol.69
, pp. 604-613
-
-
Vanhoutte, D.1
Schellings, M.2
Pinto, Y.3
|