BTLA expression contributes to septic morbidity and mortality by inducing innate inflammatory cell dysfunction

Journal of Leukocyte Biology

Volumn 92, Issue 3, 2012, Pages 593-603

  Shubin, Nicholas J ^a   Chung, Chun S ^a   Heffernan, Daithi S ^a   Irwin, Lydea R ^a   Monaghan, Sean F ^a   Ayala, Alfred ^a  

^a BROWN UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

B AND T LYMPHOCYTE ATTENUATOR; CD11B ANTIGEN; CD69 ANTIGEN; CXCL3 CHEMOKINE; HERPESVIRUS ENTRY MEDIATOR LIGAND; INTERLEUKIN 10; MAJOR HISTOCOMPATIBILITY ANTIGEN CLASS 2; MONOCYTE CHEMOTACTIC PROTEIN 1; PROGRAMMED DEATH 1 RECEPTOR; PROTEIN; UNCLASSIFIED DRUG;

ADULT; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANTIGEN PRESENTATION; ARTICLE; BACTERIAL LOAD; BONE MARROW; BONE MARROW CELL; CELL INTERACTION; CELL MATURATION; CELL POPULATION; CLINICAL ARTICLE; CONTROLLED STUDY; CRITICALLY ILL PATIENT; CYTOKINE RELEASE; DENDRITIC CELL; FEMALE; HUMAN; IMMUNE RESPONSE; INFLAMMATORY CELL; INNATE IMMUNITY; LEUKOCYTE ACTIVATION; MACROPHAGE; MALE; MONOCYTE; MORBIDITY; MORTALITY; MOUSE; NEUTROPHIL; NONHUMAN; PERITONEUM; PERITONEUM LAVAGE; PRIORITY JOURNAL; PROTEIN EXPRESSION; RENAL PROTECTION; SEPSIS; SURVIVAL;

ANIMALS; DISEASE MODELS, ANIMAL; FLOW CYTOMETRY; HUMANS; IMMUNITY, INNATE; INFLAMMATION; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MYELOID CELLS; PHENOTYPE; RECEPTORS, IMMUNOLOGIC; RECEPTORS, TUMOR NECROSIS FACTOR, MEMBER 14; SEPSIS;

BACTERIA (MICROORGANISMS); MUS;

EID: 84865457276     PISSN: 07415400     EISSN: None     Source Type: Journal    
DOI: 10.1189/jlb.1211641     Document Type: Article

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