Aryl hydrocarbon receptor protects lung adenocarcinoma cells against cigarette sidestream smoke particulates-induced oxidative stress

Toxicology and Applied Pharmacology

Volumn 259, Issue 3, 2012, Pages 293-301

  Cheng, Ya Hsin ^a   Huang, Su Chin ^b   Lin, Chun Ju ^b   Cheng, Li Chuan ^b   Li, Lih Ann ^b  

^a CHINA MEDICAL UNIVERSITY   (Taiwan)

^b NATIONAL HEALTH RESEARCH INSTITUTES   (Taiwan)

Author keywords

AhR; Antioxidant defense; Cigarette sidestream smoke particulates; Lung adenocarcinoma; Oxidative stress

Indexed keywords

AROMATIC HYDROCARBON RECEPTOR; BETA ACTIN; CIGARETTE SIDESTREAM SMOKE PARTICULATE; CIGARETTE SMOKE; CYCLIN DEPENDENT KINASE INHIBITOR 2A; CYCLIN DEPENDENT KINASE INHIBITOR 2B; CYTOCHROME P450 1B1; GLUCURONOSYLTRANSFERASE 1A10; GLUCURONOSYLTRANSFERASE 1A8; LIGAND; MESSENGER RNA; PROTEIN P21; PROTEIN P53; REACTIVE OXYGEN METABOLITE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE (PHOSPHATE) DEHYDROGENASE (QUINONE); TRANSCRIPTION FACTOR NRF2; UNCLASSIFIED DRUG;

ARTICLE; CANCER CELL CULTURE; CARCINOMA CELL; CELL CYCLE ARREST; CELL CYCLE G0 PHASE; CELL CYCLE G1 PHASE; CELL CYCLE PROGRESSION; CELL GROWTH; CELL PROLIFERATION; CELL PROTECTION; CELL VIABILITY; CLONOGENESIS; CONTROLLED STUDY; DNA DAMAGE; DNA REPAIR; GENE OVEREXPRESSION; GENE SILENCING; HUMAN; HUMAN CELL; HUMAN TISSUE; LUNG ADENOCARCINOMA; OXIDATIVE STRESS; PARTICULATE MATTER; PROTEIN EXPRESSION; SENSOR; SIGNAL TRANSDUCTION;

ADENOCARCINOMA; ARYL HYDROCARBON HYDROXYLASES; CELL LINE, TUMOR; CELL PROLIFERATION; DNA DAMAGE; G0 PHASE; G1 PHASE CELL CYCLE CHECKPOINTS; GENE EXPRESSION REGULATION, NEOPLASTIC; GENE KNOCKDOWN TECHNIQUES; GLUCURONOSYLTRANSFERASE; HUMANS; LUNG NEOPLASMS; OXIDATIVE STRESS; REACTIVE OXYGEN SPECIES; RECEPTORS, ARYL HYDROCARBON; SMOKE; TOBACCO SMOKE POLLUTION; XENOBIOTICS;

EID: 84862822849     PISSN: 0041008X     EISSN: 10960333     Source Type: Journal    
DOI: 10.1016/j.taap.2012.01.005     Document Type: Article

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