CREM α overexpression decreases IL-2 production, induces a T H17 phenotype and accelerates autoimmunity

Journal of Molecular Cell Biology

Volumn 4, Issue 2, 2012, Pages 121-123

  Lippe, Ralph ^a   Ohl, Kim ^b   Varga, Georg ^a   Rauen, Thomas ^b,c   Crispin, Jose C ^c   Juang, Yuang Taung ^c   Kuerten, Stefanie ^d   Tacke, Frank ^b   Wolf, Marc ^a   Roebrock, Kirsten ^a   Vogl, Thomas ^a   Verjans, Eva ^b   Honke, Nora ^b   Ehrchen, Jan ^a   Foell, Dirk ^a   Skryabin, Boris ^a   Wagner, Norbert ^b   Tsokos, George C ^c   Roth, Johannes ^a   Tenbrock, Klaus ^a,b  

^a UNIVERSITY OF MÜNSTER   (Germany)

^b RWTH AACHEN UNIVERSITY   (Germany)

^c BETH ISRAEL DEACONESS MEDICAL CENTER   (United States)

^d UNIVERSITY OF COLOGNE   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

CYCLIC AMP RESPONSIVE ELEMENT MODULATOR ALPHA; INTERLEUKIN 17; INTERLEUKIN 2; INTERLEUKIN 21; INTERLEUKIN 22; INTERLEUKIN 23; MESSENGER RNA; NUCLEIC ACID BINDING PROTEIN; TRANSCRIPTION FACTOR; UNCLASSIFIED DRUG;

ANIMAL EXPERIMENT; AUTOIMMUNITY; CELL LINEAGE; CYTOKINE PRODUCTION; GENE DELETION; GENE OVEREXPRESSION; IN VITRO STUDY; IN VIVO STUDY; LETTER; LYMPHOCYTE PROLIFERATION; LYMPHOCYTE SUBPOPULATION; MOUSE; NONHUMAN; PHENOTYPE; PROTEIN EXPRESSION; SYSTEMIC LUPUS ERYTHEMATOSUS; TH17 CELL; TRANSGENIC MOUSE;

ANIMALS; AUTOIMMUNITY; CYCLIC AMP RESPONSE ELEMENT MODULATOR; DOWN-REGULATION; GENE EXPRESSION; HUMANS; INTERLEUKIN-17; INTERLEUKIN-2; MICE; MICE, KNOCKOUT; TH17 CELLS;

EID: 84860210498     PISSN: 16742788     EISSN: 17594685     Source Type: Journal    
DOI: 10.1093/jmcb/mjs004     Document Type: Letter

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