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Volumn 119, Issue 7, 2012, Pages 1791-1792

The FLT3 and Pim kinases inhibitor SGI-1776 preferentially target FLT3-ITD AML cells

Author keywords

[No Author keywords available]

Indexed keywords

ACTIN; CASPASE 3; FLT3 LIGAND; LIPOCORTIN 5; MITOGEN ACTIVATED PROTEIN KINASE; NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE; PROTEIN KINASE B; PROTEIN PIM; PROTEIN SERINE THREONINE KINASE; PROTEIN SERINE THREONINE KINASE INHIBITOR; QUIZARTINIB; SGI 1776; STAT5 PROTEIN; UNCLASSIFIED DRUG;

EID: 84857493456     PISSN: 00064971     EISSN: 15280020     Source Type: Journal    
DOI: 10.1182/blood-2011-11-393066     Document Type: Letter
Times cited : (35)

References (9)
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  • 2
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    • Pim kinase inhibitor, SGI-1776, induces apoptosis in chronic lymphocytic leukemia cells
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  • 3
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    • Mechanisms of cytotoxicity to Pim kinase inhibitor, SGI-1776, in acute myeloid leukemia
    • Chen LS, Redkar S, Taverna P, Cortes JE, Gandhi V. Mechanisms of cytotoxicity to Pim kinase inhibitor, SGI-1776, in acute myeloid leukemia. Blood. 2011; 118(3):693-702.
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  • 4
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  • 5
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    • AC220 is a uniquely potent and selective inhibitor of FLT3 for the treatment of acute myeloid leukemia (AML)
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  • 8
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    • Protein synthesis is resistant to rapamycin and constitutes a promising therapeutic target in acute myeloid leukemia
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.