Prevention of neonatal oxygen-induced brain damage by reduction of intrinsic apoptosis

Cell Death and Disease

Volumn 3, Issue 1, 2012, Pages

  Sifringer, M ^a   Bendix, I ^b   Borner C ^a   Endesfelder, S ^a   Von Haefen, C ^a   Kalb, A ^a   Holifanjaniaina, S ^c,d   Prager, S ^b   Schlager, G W ^b   Keller, M ^b   Jacotot, E ^c,d,e   Felderhoff Mueser, U ^b  

^a CHARITÉ UNIVERSITÄTSMEDIZIN BERLIN   (Germany)

^b UNIVERSITY HOSPITAL ESSEN   (Germany)

^c HÔPITAL ROBERT DEBRÉ   (France)

^d UNIVERSITÉ PARIS DESCARTES   (France)

^e HAMMERSMITH HOSPITAL   (United Kingdom)

Author keywords

Brain damage; Caspase inhibitor; Hyperoxia; Neonatal cell death; Neuroprotection

Indexed keywords

APOPTOSIS INDUCING FACTOR; CASPASE 2; CASPASE INHIBITOR; CYTOCHROME C; OXYGEN; TRP 601; UNCLASSIFIED DRUG; APOPTOTIC PROTEASE ACTIVATING FACTOR 1; BID PROTEIN, HUMAN; CASP2 PROTEIN, HUMAN; CASPASE 3; CYSTEINE PROTEINASE; NEUROPROTECTIVE AGENT; OLIGOPEPTIDE; PROTEIN BID; QUINOLIN 2 CARBONYL VD(OME)VAD(OME) CH2 O(2,6F2)PH; QUINOLIN-2-CARBONYL-VD(OME)VAD(OME)-CH2-O(2,6F2)PH; QUINOLINE DERIVATIVE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; BRAIN DAMAGE; ENZYME ACTIVITY; HYPEROXIA; NEUROPROTECTION; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT; ANIMAL; BRAIN; BRAIN INJURY; DRUG ANTAGONISM; DRUG EFFECT; GENE EXPRESSION REGULATION; GENETICS; HUMAN; INFANT; METABOLISM; NEWBORN; PATHOLOGY; PREMATURITY; SIGNAL TRANSDUCTION; WISTAR RAT;

RODENTIA;

ANIMALS; ANIMALS, NEWBORN; APOPTOSIS; APOPTOTIC PROTEASE-ACTIVATING FACTOR 1; BH3 INTERACTING DOMAIN DEATH AGONIST PROTEIN; BRAIN; BRAIN INJURY, CHRONIC; CASPASE 2; CASPASE 3; CYSTEINE ENDOPEPTIDASES; CYTOCHROMES C; GENE EXPRESSION REGULATION; HUMANS; HYPEROXIA; INFANT; NEUROPROTECTIVE AGENTS; OLIGOPEPTIDES; OXYGEN; PREMATURE BIRTH; QUINOLINES; RATS; RATS, WISTAR; SIGNAL TRANSDUCTION;

EID: 84856943764     PISSN: None     EISSN: 20414889     Source Type: Journal    
DOI: 10.1038/cddis.2011.133     Document Type: Article

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