Iodide treatment acutely increases pendrin (SLC26A4) mRNA expression in the rat thyroid and the PCCl3 thyroid cell line by transcriptional mechanisms

Molecular and Cellular Endocrinology

Volumn 350, Issue 1, 2012, Pages 118-124

  Calil Silveira, Jamile ^a   Serrano Nascimento, Caroline ^a   Nunes, Maria Tereza ^a  

^a UNIVERSITY OF SÃO PAULO   (Brazil)

Author keywords

Escape phenomenon; Iodide; Pendrin; Perchlorate; SLC26A4; Wolff Chaikoff effect

Indexed keywords

DACTINOMYCIN; IODIDE; MESSENGER RNA; SODIUM PERCHLORATE; THIAMAZOLE;

ANIMAL CELL; ANIMAL CELL CULTURE; ANIMAL EXPERIMENT; ARTICLE; CELL ASSAY; CELL COUNT; CELL VIABILITY; CONTROLLED STUDY; GENE; GENE EXPRESSION REGULATION; GENE LOCATION; HYPOPHYSIS THYROID SYSTEM; MALE; NONHUMAN; PENDRIN GENE; PRIORITY JOURNAL; RAT; THYROID FUNCTION; TRANSCRIPTION REGULATION; UPREGULATION;

ANIMALS; ANTITHYROID AGENTS; CELL LINE; CELL PROLIFERATION; CELL SURVIVAL; CHLORIDE-BICARBONATE ANTIPORTERS; DACTINOMYCIN; IODIDES; MALE; METHIMAZOLE; PERCHLORIC ACID; PITUITARY GLAND; PROTEIN SYNTHESIS INHIBITORS; RATS; RATS, WISTAR; RNA, MESSENGER; THYROID GLAND; THYROTROPIN; THYROXINE; TRANSCRIPTION, GENETIC; TRIIODOTHYRONINE;

EID: 84856046096     PISSN: 03037207     EISSN: 18728057     Source Type: Journal    
DOI: 10.1016/j.mce.2011.12.002     Document Type: Article

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