Rhomboid-7 over-expression results in Opa1-like processing and malfunctioning mitochondria

Biochemical and Biophysical Research Communications

Volumn 414, Issue 2, 2011, Pages 315-320

  Rahman, Mokhlasur ^a,b   Kylsten, Per ^b  

^a UNIVERSITY OF TEXAS SOUTHWESTERN MEDICAL CENTER   (United States)

^b SÖDERTÖRN UNIVERSITY   (Sweden)

Author keywords

Mitochondria; Opa1; Rhomboid 7; Synapse

Indexed keywords

ADENOSINE TRIPHOSPHATE; CYTOCHROME C OXIDASE; PEPTIDES AND PROTEINS; PROTEIN OPA1; RHOMBOID 7; UNCLASSIFIED DRUG;

APOPTOSIS; ARTICLE; CELL AGGREGATION; CELLULAR DISTRIBUTION; CENTRAL NERVOUS SYSTEM; DROSOPHILA MELANOGASTER; GENE OVEREXPRESSION; LETHALITY; LIFESPAN; LOCOMOTION; MITOCHONDRION; NERVE CELL; NEUROMUSCULAR FUNCTION; NONHUMAN; PRIORITY JOURNAL;

ADENOSINE TRIPHOSPHATE; ANIMALS; APOPTOSIS; DROSOPHILA MELANOGASTER; DROSOPHILA PROTEINS; ELECTRON TRANSPORT COMPLEX IV; EYE; LONGEVITY; MEMBRANE PROTEINS; MITOCHONDRIA; MITOCHONDRIAL PROTEINS; NEUROMUSCULAR JUNCTION; NEURONS;

ANIMALIA; DROSOPHILA MELANOGASTER;

EID: 80054841370     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2011.09.047     Document Type: Article

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