Rottlerin increases cardiac contractile performance and coronary perfusion through BKca++ channel activation after cold cardioplegic arrest in isolated hearts

Circulation

Volumn 124, Issue 11 SUPPL. 1, 2011, Pages

  Clements, Richard T ^a   Cordeiro, Brenda ^a   Feng, Jun ^a   Bianchi, Cesario ^a   Sellke, Frank W ^a  

^a BROWN UNIVERSITY   (United States)

Author keywords

Akt; cardioplegia; ischemia; potassium channels; protein kinase C; rottlerin

Indexed keywords

LARGE CONDUCTANCE CALCIUM ACTIVATED POTASSIUM CHANNEL; PAXILLINE; ROTTLERIN;

ANIMAL CELL; ARTICLE; CARDIOPLEGIA; CARDIOPULMONARY BYPASS; CONTROLLED STUDY; CORONARY ARTERY BLOOD FLOW; HEART LEFT VENTRICLE ENDDIASTOLIC PRESSURE; HEART MUSCLE CELL; HEART MUSCLE CONTRACTILITY; HEART PROTECTION; ISOLATED HEART; MALE; NONHUMAN; PRIORITY JOURNAL; RAT;

ACETOPHENONES; ANIMALS; BENZOPYRANS; COLD TEMPERATURE; CORONARY VESSELS; ENZYME INHIBITORS; HEART ARREST, INDUCED; INDOLES; MALE; MODELS, ANIMAL; MYOCARDIAL CONTRACTION; MYOCYTES, CARDIAC; POTASSIUM CHANNEL BLOCKERS; POTASSIUM CHANNELS; PROTEIN KINASE C-DELTA; PROTO-ONCOGENE PROTEINS C-AKT; RATS; RATS, SPRAGUE-DAWLEY;

EID: 80052717086     PISSN: 00097322     EISSN: 15244539     Source Type: Journal    
DOI: 10.1161/CIRCULATIONAHA.110.012112     Document Type: Article

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