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Volumn 129, Issue 3, 2011, Pages 1019-1020

RAD51C germline mutations in Chinese women with familial breast cancer

Author keywords

[No Author keywords available]

Indexed keywords

AMINO ACID SUBSTITUTION; BREAST CANCER; CANCER SUSCEPTIBILITY; CELL CYCLE ARREST; CHINESE; CHROMOSOME 17Q; DNA DAMAGE; EXON; FAMILIAL DISEASE; GENE MUTATION; GENE SEQUENCE; GENETIC SUSCEPTIBILITY; GENETIC VARIABILITY; HOMOLOGOUS RECOMBINATION; HUMAN; INTRON; LETTER; ONCOGENE; POLYMERASE CHAIN REACTION; PRIORITY JOURNAL; RAD51C GENE; TUMOR SUPPRESSOR GENE;

EID: 80052601847     PISSN: 01676806     EISSN: 15737217     Source Type: Journal    
DOI: 10.1007/s10549-011-1574-3     Document Type: Letter
Times cited : (26)

References (10)
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  • 3
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  • 5
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    • RAD51C Is Required for Holliday Junction Processing in Mammalian Cells
    • DOI 10.1126/science.1093037
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  • 6
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    • Resolving RAD51C function in late stages of homologous recombination
    • DOI 10.1186/1747-1028-2-15
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    • RAD51C facilitates checkpoint signaling by promoting CHK2 phosphorylation
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.